[1. Adams DR, Victor M. Principles of Neurology. New York, Mc Graw-Hill Inc, 1993.]Search in Google Scholar
[2. Skidmore CT, Andrefsky J. Spontaneous intracerebral hemorrhage: epidemiology, patophysiology, and medical menagement. Neurosurg Clin N Am. 2002; 13: 281–288.10.1016/S1042-3680(02)00019-0]Search in Google Scholar
[3. Bahemuka M, Primary intracerebral hemorrhagy and heart weight: a clinicopathological case control re view of 218 patients. Stroke. 1987; 18: 531–536.10.1161/01.STR.18.2.5313564115]Search in Google Scholar
[4. Huang FP, Xi G, Keep RF, et al. Brain edema after experimental intracerebral hemorrage; Role of hemglobin degradation products. J Neurosurg. 2002; 96: 287–293.10.3171/jns.2002.96.2.028711838803]Search in Google Scholar
[5. Nagase H. Activation mechanisms of matrix metaloproteinases. Biol Chem. 1997; 378: 151–160.]Search in Google Scholar
[6. Rosenberg G, Navratil M. Metalloproteinase inhbition blocks edema in intracerebral hemorrhage in the rat. Neurology. 1997; 48: 921–926.10.1212/WNL.48.4.921]Search in Google Scholar
[7. Lapchak P, Chapman D, Zivin J. Metalloproteinase inhibition reduces thrombolytic (tissue plasminogen activator)-induced hemorrhage after thromboembolic stroke. Stroke. 2000; 31: 3034–40.10.1161/01.STR.31.12.3034]Search in Google Scholar
[8. Pfefferkorn T, Rosenberg G. Closure of the blood– brain barrier by matrix metalloproteinase inhibition reduces rtPA-mediated mortality in cerebral ischemia with delayed reperfusion. Stroke. 2003; 34: 2025–30.10.1161/01.STR.0000083051.93319.2812855824]Search in Google Scholar
[9. Alvarez-Sabin J, Delgado P, Abilleira S, Molina C, Arenillas J, Ribo M, et al. Temporal profile of matrix metalloproteinases and their inhibitors after spontneous intracerebral hemorrhage: relationship to clincal and radiological outcome. Stroke. 2004; 35: 1316–22.10.1161/01.STR.0000126827.69286.9015087562]Search in Google Scholar
[10. Abilleira S, Montaner J, Molin C, et al. Matrix metaloproteinase-9 contretation after spontaneous intracrebral hemorrhage. J Neurosurg. 2003; 99: 65–70.10.3171/jns.2003.99.1.006512854746]Search in Google Scholar
[11. De Graba JT, Hallenbeck MJ, Pettigrew DK, et al. Progresion in acute stroke: Value of the initial NIH stroke scale score on patients stratification in future trials. Stroke. 1999; 30: 1208–1212.10.1161/01.STR.30.6.120810356101]Search in Google Scholar
[12. Baird AE, Dasche J, Connor A, et al. Comparision of retrospective and prospecive measurements of the national institutes of health stroke scale. Cerebrovasc Dis. 2000; 10: 80–81.10.1159/00001603110629353]Search in Google Scholar
[14. Adams H. Handbook of cerebrovascular disease. New York: Dekker; 1996]Search in Google Scholar
[15. Abilleira S, Montaner J, Molin C, et al. Matrix metalloproteinase-9 contretation after spontaneous intracerebral hemorrhage. J Neurosurg 2003; 99: 65–70.10.3171/jns.2003.99.1.006512854746]Search in Google Scholar
[16. Leira R, Davalos A, Silva Y, et al. Stroke Project, Cerebrovascularis Diseases Group of Spanish Neu rological Society. Early neurological deterioration in intracerebral hemorrage: predictors and associated factors. Neurology. 2004; 63: 461–467.10.1212/01.WNL.0000133204.81153.AC15304576]Search in Google Scholar
[17. Tejima E, Guo S, Murata Y, et al. Neuroprotective effects of overexpressing tissue inhibitor of metalloproteinase TIMP-1. J Neurotrauma. 2009; 26(11): 1935–1941.10.1089/neu.2009.0959282280419469687]Search in Google Scholar
[18. Mahajan SD, Aalinkeel R, Reynolds JL, et al. Suppression of MMP-9 expression in brain micro-vascular endothelial cells (BMVEC) using a gold nanorod (GNR)-siRNA nanoplex. Immunol Invest. 2012; 41(4): 337–355.10.3109/08820139.2011.60486321864113]Search in Google Scholar