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Fetal Echocardiography in Uncommon Prenatal Cardiac Anomalies: Right Atrium Diverticulum, Interventricular Septal Aneurysm, Left and Right Ventricle Diverticulum – Report from Referral Center for Fetal Cardiology in Poland

. Therapeutic use of heparin beyond anticoagu- lation. Current Drug Discovery Technologies 2009, 6, 4, 281-289 10. Young E. The anti-inflammatory effects of heparin and related compounds. Thrombosis Research, vol. 122, no. 6, pp. 743-752, 2008. 11. Respondek-Liberska M, Sokołowski Ł, Słodki M, Zych-Krekora K, Strzelecka I, Krekora M, Maroszyńska I, Moll J, Moll J. Prenatal diagnosis of TAPVC on Monday, delivery of Tuesday and cardiac surgery at Wednsday - a model of perinatal care in 3rd trimester in case of fetal/neonatal critical heart defect

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What is the Survival Rate in Prenatally Detected Cantrell’s Pentalogy?

References 1. Cantrell JR, Haller JA, Ravitch MM. A syndrome of congenital defects involving the abdominal wall, sternum, diaphragm, pericardium, and heart. Surg Gynecol Obstet. 1958;107:602–614. 2. Desselle C, Herve P, Toutain A, Lardy H, Sembely C, Perrotin F. Pentalogy of Cantrell: sonographic assessment. J Clin Ultrasound. 2007;35:216–220. 3. Khoury MJ, Cordero JF, Rasmussen S. Ectopia cordis, midline defects and chromosome abnormalities: an epidemiologic perspective. Am J Med Genet. 1988;30:811–817. 4. Respondek-Liberska M, Janiak K

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Constriction of Fetal Ductus Arteriosus and Maternal Intake of Polyphenol-Rich Foods

, 89 (4): 251-6 24. Coceani F et al.: Deletion of the endothelin-A-receptor suppresses oxygeninduced constriction but not postnatal closure of the ductus arteriosus. J Cardiovasc Pharmacol, 2000. 36 (5 Suppl 1): S75-7 25. Baragatti B, et al.: Cytochrome P-450 3A13 and endothelin jointly mediate ductus arteriosus constriction to oxygen in mice. Am J Physiol Heart Circ Physiol 2011, 300 (3): H892-901 26. Momma KT, Nakanishi and Imamura S. .: Inhibition of in vivo constriction of fetal ductus arteriosus by endothelin

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Fatty Acid Metabolism Disorder as a Factor in Atherogenesis

76(6): 1222-1229, 2002. 14. Frattola L, Canal N, Andreoli VM, Maffei F, Tonon GC. Lipid changes in neurogenic muscular atrophies. Research in experimental medicine 164(3): 241-246, 1974. 15. Julien C, Berthiaume L, Hadj-Tahar A, et al. Postmortem brain fatty acid profile of levodopa-treated Parkinson disease patients and parkinsonian monkeys. Neurochem Int 48(5): 404-414, 2006. 16. Hamlat N, Forcheron F, Negazzi S, et al. Lipogenesis in arterial wall and vascular smooth muscular cells: regulation and abnormalities in insulin

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Ultrasound elastography can detect placental tissue abnormalities

artery Doppler measurement; amniotic fluid volume, and fetal heart rate monitoring) may facilitate the identification of SGA due to placental insufficiency. Indeed, Karaman et al . described a putative association between the uterine artery pulsatility index and elastographic indices of the placenta. 30 Another limitation of this study is that strain elastography requires manual compression with the examiner’s hands, although it has been reported that reproducible data can be obtained when a single examiner conducts the examination. 31 Furthermore, only placentas

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Retrospection of the effect of hydroxyurea treatment in patients with sickle cell disease

patients [ 10 ]. Further, these patients – in younger age – have increased susceptibility to infections, acute chest syndrome, and stroke, while – in older age – they are susceptible to retinopathy, as well as damage to the lungs, kidney, and heart [ 11 , 12 ]. In addition to VOC, sickle cell patients experience sequestration crisis (pooling of blood in an organ), aplastic crisis (reduced function of bone marrow), and hemolytic crisis (rapid breakdown of blood cells). Presence of high levels of fetal hemoglobin (HbF) inhibits polymerization in SCA patients, highlighting

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Congenital abdominal aortic aneurysm in a term neonate: a case report

Congenital abdominal aortic aneurysm (AAA), a rare condition in neonates, is usually associated with umbilical artery catheterization [ 1 , 2 ] or other conditions such as congenital heart or aortic malformation, infection (mycotic aneurysm) [ 3 , 4 ], connective tissue diseases (Marfan syndrome, Ehlers–Danlos syndrome, Loeys–Dietz syndrome) [ 5 , 6 ], or vasculitis (Takayasu’s disease, Kawasaki syndrome) [ 7 ]. An idiopathic congenital AAA is extremely rare. We report a case of idiopathic congenital AAA consisting of multiple lesions in a Thai neonate born

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Evaluation of intestinal damage biomarkers in calves with atresia coli

, and the cause of the abnormality is found to be the presence of homozygous recessive alleles of the defective gene for atresia coli ( 5 , 28 ). Vascular insufficiency of the developing colon in calves and diagnosis during early pregnancy in cattle by amniotic palpation may damage the foetal intestinal blood supply and disrupt organogenesis, thereby increasing the risk of atresia coli ( 5 ). Mechanical obstructions are a result of abnormalities in the bowel lumen, intestinal wall, or outside the intestinal tract. Congenital intestinal obstructions in calves, such

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Critical Care Aspects of Gallstone Disease

surface (b), multiple stones (c). The cut surface is shown with a brownish centre and radial disposition of cholesterol monohydrate crystals (d). Pure pigment stones are shown as debris (e). Mixed stones are shown as multifaceted concernments (f). Note that the black pigment surface and the inner centre are made of a small amount of solid cholesterol crystals (arrow) Black horizontal line=1 cm. The rising prevalence and incidence of cholesterol gallstones parallel the concomitant rise of several metabolic abnormalities, such as insulin resistance and type 2

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NPS2390, a selective calcium-sensing receptor antagonist controls the phenotypic modulation of hypoxic human pulmonary arterial smooth muscle cells by regulating autophagy

Introduction The hypoxic pulmonary hypertension (HPAH) is a clinical syndrome caused from increasing pulmonary vascular resistance, which can cause serious impairment of heart and lung function. Hypoxic pulmonary vascular remodeling (HPVR) is an important pathologic basis, but the exact mechanisms have not been fully elucidated. The abnormal proliferation of pulmonary arterial smooth muscle cells (PASMCs) play an important role during this process. Our previous study have found that phenotypic modulation of PASMCs is a key step in initiating HPVR, but the

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