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Cardiogenic shock after nifedipine administration in a pregnant patient: A case report and review of the literature

effects than betamimetics. [ 1 , 2 , 3 , 4 , 5 ] Maternal serious adverse events have been described in case reports. [ 6 , 7 , 8 , 9 , 10 , 11 ] We present the case of a 21-year-old woman who developed a severe cardiogenic choc after the administration of nifedipine. Case Report A 21-year-old Caucasian woman at 27 weeks of pregnancy was admitted to the obstetric department for pre-term labor. She received 10 mg of nifedipine 4 times in 1 h, according to the internal protocol. Shortly after, she brutally deteriorated with pulmonary edema and hypoxemia

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Assessment of Respiratory Complications Associated with End Stage Renal Disease in Northern Saudi Arabia

. Uremic pleuritis - a clinico-pathological entity. N Engl J Med 1969; 281(5):255-6. 10. MCMURRAY SD, LUFT FC, MAXWELL DR, HAMBURGER RJ, FUTTY D, SZWED JJ, et al. Prevailing patterns and predictor variables in patients with acute tubular necrosis. Arch Intern Med 1978; 138(6):950-5. 11. SARNAK MJ, JABER BL. Pulmonary infectious mortality among patients with end-stage renal disease. Chest 2001; 120(6):1883-7. 12. HALLE MP, HERTIG A, KENGNE AP, ASHUNTANTANG G, RONDEAU E, RIDEL C. Acute pulmonary oedema in chronic dialysis

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Acute Respiratory Distress Syndrome Caused by Methadone Syrup

9. Drudi FM, Poggi R, Trenta F, Manganaro F, Iannicelli E. [A case of the adult respiratory distress syndrome induced by a methadone overdose, in Italian]. Radiol Med 1997;94:393-6. PMID: 9465250 10. Schaaf JT, Spivack ML, Rath GS, Snider GL. Pulmonary edema and adult respiratory distress syndrome following methadone abuse. Am Rev Respir Dis 1973;107:1047-51. PMID: 4740265 11. Martin M, Daoudal P, Bein C, Bazzi N, Bourscheid D, Floriot C. [Inhaled methadone-induced non cardiogenic pulmonary edema with Acute Respiratory Distress

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Investigation on Factors Associated with Severe Hand, Foot and Mouth Disease

al. Clinical feature and risk factors of pulmonary oedema after enterovirus-71-related hand, foot, and mouth disease. Lancet 1999; 354:1682-1686. 7. Chen HI, Liao JF, Kuo L, Ho ST. Centrogenic pulmonary edema induced by cerebral compression in rats. Circ Res 1980; 47:366-373. 8. Kao SJ, Yang FL, Hsu YH, Chen HI. Mechanism of fulminant pulmonary edema caused by enterovirus 71. Clin Infect Dis 2004;38(12):1784-1788. 9. Chan LG, Parashar UD, Lye MS, Ong FG, Zaki SR, Alexander JP, et al. Deaths of children during an

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Pulmonary veno-occlusive disease: a rare cause of pulmonary hypertension in systemic sclerosis. Case presentation and review of the literature

pulmonary arterial hypertension associated with connective tissue diseases . Hum Pathol 2007; 38 : 893–902. 26. NICOD P, MOSER KM. Primary pulmonary hypertension. The risk and benefit of lung biopsy. Circulation.1989; 80 (5): 1486-1488. 27. PALMER SM, ROBINSON LJ, WANG A, GOSSAGE JR, BASHORE T, TAPSON VF. Massive pulmonary oedema and death after prostacyclin infusion in a patient with pulmonary venoocclusive disease . Chest 1998; 113 : 237–240. 28. CREAGH-BROWN BC, NICHOLSON AG, SHOWKATHALI R, GIBBS JS, HOWARD LS. Pulmonary veno-occlusive disease

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Renovascular Hypertension: Clinical Features, Differential Diagnoses and Basic Principles of Treatment

Kidney. Clarkson MR, Magee CN, Brenner BM. Eds. Philadelphia: Oxford University Press 2010: 468-86. 10. Adamczak M, Wiecek A. Ischemic nephropathy - pathogenesis and treatment. Nefrologia 2012; 32(4): 432-8. 11. Messerli FH, Bangalore S, Makani H, Rimoldi SF, Allemann Y, White CJ, Textor S, Sleight P. Flash pulmonary oedema and bilateral renal artery stenosis: the Pickering Syndrome. Eur Heart J 2011; 32(18): 2231-7. 12. Petrović D. Vojinović R, Lukić S, Novaković B, Mijailović M, Jagić N. Renovascular hypertension: diagnostic and therapy. Medicus

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Electrical bioimpedance spectroscopy in time-variant systems: Is undersampling always a problem?

from the time-invariant impedance signal, all depending on the ratio between impedance estimation time and interference frequency. For wideband interferences, averaging will always reduce the effect of aliasing and the degree of attenuation will depend upon the width of the frequency distribution of the interference and on the averaging time. Case Study – Pulmonary oedema Pulmonary oedema is the accumulation of fluid in the alveoli and parenchyma of the lungs and transthoracic impedance spectroscopy (TIS) is a potential method for monitoring pulmonary oedema

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Spontaneous regression of severe aortic stenosis after massive embolization in a patient with antiphospholipid syndrome

Summary

We present a case of probably unreported aortic stenosis evolution in a patient with primary antiphospholipid syndrome (APS). A female patient, 54 years old, with a history of recurrent deep venous thrombosis, an episode of pulmonary embolism and was positive for APS antibodies. She was kept on warfarin and aspirin. The patient was admitted with acute pulmonary edema and severe aortic stenosis. While preparing for aortic valve surgery, the patient developed acute stroke, and a week later developed concurrent acute ischemia of both lower limbs. Emergency surgery salvaged the lower limbs and follow up transthoracic echocardiography showed marked regression of the thickening of the aortic valve leaflets, only mild aortic stenosis and moderate aortic regurgitation. Aortic valve surgery was cancelled, and the patient was kept on warfarin and aspirin.

Conclusion

Development of severe aortic stenosis is uncommon in primary APS, and this scenario of spontaneous improvement from a severe to a mild aortic stenosis, is probably unreported before.

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Neurogenic stress cardiomyopathy following aneurysmal subarachnoid haemorrhage: a literature review

Summary

Neurogenic stress cardiomyopathy (NSC) is defined as transient cardiac dysfunction occurring after primary brain injury, such as aneurysmal subarachnoid haemorrhage, and characterised by left ventricular systolic dysfunction with reduced ejection fraction and abnormalities of regional wall motion. It may also be suspected if elevated levels of cardiac biomarkers and ECG abnormalities are present. It is a reversible condition with favourable long-term prognosis if diagnosed and treated timely, however, NSC is associated with higher rates of early mortality and complications, including pulmonary oedema, cardiogenic shock, delayed cerebral ischaemia. Early diagnosis of the NSC is important in order to prevent these complications and reduce mortality. Management of the NSC is complicated and a multidisciplinary approach is usually required.

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Diagnosing acute respiratory distress syndrome with the Berlin definition: Which technical investigations should be the best to confirm it?

The distinction between hydrostatic pulmonary edema (HPE) and acute respiratory distress syndrome (ARDS) could be challenging in the management of acutely ill patients. The Berlin definition of acute respiratory distress syndrome (ARDS) stipulate that respiratory failure must not be explained by cardiac failure of fluid overload. The situation needs an objective assessment to exclude hydrostatic edema, if no risk factor is present. [ 1 ] The differential diagnosis is based on the evaluation of the left atrial pressure, which is considered as normal in ARDS

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