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References 1. Sogolow S.R. An historical review of the use of oxytocin prior to delivery. ObstetGynecolSurv. 1966; 21(2):155–72 2. Olff M., Frijling J. L., Kubzansky M., et al. The role of oxytocin in social bonding, stress regulation and mental health: an update on the moderating effects of context and interindividual differences. Psychoneuroendocrinology, 2013; 38(9), 1883-1894 3. Tachibana M., et al. Long-term administration of intranasal oxytocin is a safe and promising therapy for early adolescent boys with autism spectrum disorders. Journal of child and ado

gene leads to deficits in synaptic function, social interaction, and social communication. Mol Autism 1, 15, 2010. Brambilla P, Hardan A, di Nemi SU, Perez J, Soares JC, Barale F. Brain anatomy and development in autism: review of structural MRI studies. Brain Res Bull 61, 557–569, 2003. Carper RA, Courchesne E. Localized enlargement of the frontal cortex in early autism. Biol Psychiatry 57, 126–133, 2005. Chapman DB, Theodosis DT, Montagnese C, Poulain DA, Morris JF. Osmotic stimulation causes structural plasticity of neurone-glia relationships of the oxytocin but

References Akman T, Akman L, Erbas O, Terek MC, Taskiran D, Ozsaran A. The preventive effect of oxytocin to cisplatin-induced neurotoxicity: an experimental rat model. Biomed Res Int 2015, 167235, 2015. Bakos J, Strbak V, Ratulovska N, Bacova Z. Effect of oxytocin on neuroblastoma cell viability and growth. Cell Mol Neurobiol 32, 891–896, 2012. Cargnello M, Roux PP. Activation and function of the MAPKs and their substrates, the MAPK-activated protein kinases. Microbiol Mol Biol Rev 75, 50–83, 2011. Cassoni P, Sapino A, Stella A, Fortunati N, Bussolati G. Presence

cyclinD1 and p60-katanin increase in rat hippocampal neurons. Eur J Neurosci 37, 1610-1619, 2013. Krug AK, Balmer NV, Matt F, Schonenberger F, Merhof D, Leist M. Evaluation of a human neurite growth assay as specific screen for developmental neurotoxicants. Arch Toxicol 87, 2215-3221, 2013. Lestanova Z, Bacova Z, Kiss A, Havranek T, Strbak V, Bakos J. Oxytocin Increases Neurite Length and Expression of Cytoskeletal Proteins Associated with Neuronal Growth. J Mol Neurosci 2015. [Epub ahead of print]. Lee SH, Dominguez R. Regulation of actin cytoskeleton dynamics in cells

References Barker SB, Dawson KS (1998): The effects of animal-assisted therapy on anxiety ratings of hospitalized psychiatric patients. Psychiatric Services, 49, 797-802. Beetz A, Uvnäs-Moberg K, Julius H, Kotrschal K (2012): Psychosocial and psychophysiological effects of human- animal interactions: the possible role of oxytocin. Frontiers in Psychology, 3, 1-15. doi: 10.3389/fpsyg.2012.00234. Benešová M, Zouharová M (2007): Methodically described procedures and specified activities (techniques) of canistherapy. In: Veleminský M. (ed.): Zootherapy in light of

phosphodiesterases and the contractile activity in human pregnant myometrium near term. Biochem Pharmacol 1989; 38: 9-15. Franova S, Janicek F, Visnovsky J, Dokus K, Zubor P, Sutovska M, Nosalova G. Utero-relaxant effect of PDE4-selective inhibitor alone and in simultaneous administration with 2-mimetic on oxytocin-induced contrations in pregnant myometrium. J Obstet Gynaecol Res 2009; 35: 20-25. Magraner J, Morcillo E, Ausina P, Pinto FM, Martin JD, Moreau J, Anselmi E, Barrachina MD, Cortijo J, Advenier C, Candenas ML. Effects of Mn 2+ on the responses induced by different

. Emiliano ABF, Cruz T, Pannoni V, Fudge JL. The interface of oxytocin-labeled cells and serotonin transporter-containing fibers in the primate hypothalamus: A substrate for SSRIs therapeutic effects? Neuropsychopharmacology 32, 977–988, 2007. Higashida H. Somato-axodendritic release of oxytocin into the brain due to calcium amplification is essential for social memory. J Physiol Sci 66, 275–282, 2016. Hou D, Jin F, Li J, Lian J, Liu M, Liu X, Xu Y, Zhang C, Zhao C, Jia S, Jiao R, Liu X, Wang X, Zhang Y, Wang YF. Model roles of the hypothalamo-neurohypophysial system in

foetal distress when no oxytocin is used. All other CS were classified as a form of dystocia. Sub-groups of dystocia depend on whether progress of labour (cervical dilatation) is less than 1 cm/h (inefficient uterine action) or more than 1 cm/h (efficient uterine action). Inefficient uterine action was then subdivided into poor response (despite maximum treatment with oxytocin), inability to treat adequately (for foetal reasons), inability to treat adequately (because of the uterus over-contracting), or no treatment (oxytocin not given for other reasons). Table 1

, uterine contraction characteristics (duration, frequency, and interval), and fetal heart rates. All data were measured at baseline and repeated measures at one-hour interval in addition to other birth outcomes such as mode of delivery, oxytocin use, and the duration of each stage of labor. Tool III: Apgar's score 15 at first and fifth minute to evaluate the neonatal outcomes. Tool IV: Visual analogue pain intensity scale: It is a standardized linear scale that was adopted by Mc Caffery and Pasero 16 and used to evaluate pain severity. It is a self-reported 10 cm

-263. Fort P., Salvert D., Hanriot L., Jego S., Shimizu H., Hashimoto K., Mori M., Lup- pi P.H. (2008). The satiety molecule nesfatin-1 is co-expressed with melanin concentrating hormone in tuberal hypothalamic neurons of the rat. Neuroscience, 155: 174-181. Gina L., Yosten C., Samson W.K. (2010). The anorexigenic and hypertensive effects of nesfatin-1 are reversed by pretreatment with an oxytocin receptor antagonist. Am. J. Physiol. Regul. Integr. Comp. Physiol., 298: 1642-1647. Goebel M., Stengel A., Wang L., Tach é Y. (2011). Central nesfatin-1 reduces the nocturnal