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Lead Toxicity in a Family as a Result of Occupational Exposure

References World Health Organization (WHO). Inorganic lead. Geneva: World Health Organization; Environ Health Criteria 165; 1995. Lewis J. Lead poisoning: A Historical Perspective. EPA Journal - May 1985. Hernberg S. Lead Poisoning in a Historical Perspective. Am J Ind Med 2000;38,244-54. Clark CS, Rampal KG, Thuppil V, Chen CK, Clark R, Roda S. The lead content of currently available new residential

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Impact of vitamin D receptor gene polymorphisms on blood lead levels in Thai lead exposed workers

References 1. Fullmer CS. Intestinal interactions of lead and calcium. Neurotoxicol. 1992; 13:799-807. 2. Godwin HA. The biological chemistry of lead. Curr Opin Chem Biol. 2001; 5:223-7. 3. DeLuca HF. Overview of general physiologic features and functions of vitamin D. Am J Clin Nutr. 2004; 80 (6Suppl):1689S-96S. 4. Onalaja AO, Claudio L. Genetic susceptibility to lead poisoning. Environ Health Perspect. 2000; 108 (Suppl 1):23-8. 5. Rezende VB, Barbosa F, Jr., Montenegro MF

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Lead toxicity: a review

REFERENCES Advisory Committee on Childhood Lead Poisoning Prevention (ACCLPP)”. CDC. (2012). Retrieved 19 sept. 2014. Agency for Toxic Substances and Disease Registry (ATSDR). (2007). Lead Toxicity: Who Is at Risk of Lead Exposure? Environmental Health and Medicine Education . U.S. Department of Health and Human Services. Course: WB 1105. Apostoli P, Kiss P, Porru S, Bonde JP, Vanhoorne M. (1998). Male reproductive toxicity of lead in animals and humans. ASCLEPIOS Study Group. Occup Environ Med 55 : 364–74. Barbosa Jr F, Tanus

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Toxicity of lead: a review with recent updates

its reversibility. Free Rad Biol Med 51 : 257-281. Flora SJS, Flora G, Saxena G. (2006). Environmental occurrence, health effects and management of lead poisoning. (In: Jose, S. C, Jose, S., eds. Lead . Amsterdam: Elsevier Science B.V.). pp. 158-228. Flora SJ, Pande M, Mehta A. (2003). Benefi cial eff ect of combined administration of some naturally occurring antioxidants (vitamins) and thiol chelators in the treatment of chronic lead intoxication. Chem Biol Interact 145 : 267-280. Flora SJ, Flora G, Saxena G

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Risk Of Lead Intoxication In Exposed Workers

, Tsalev D. Lead levels in blood, nails and teeth as biomarkers of exposure – informational value and relevance. Bulgarian Journal of Chemistry. 2013;2(4):133-44. Bulgarian. 8. Gordon JN. Taylor A. Bennett PN. Lead poisoning: case studies. Br J Clin Pharmacol. 2002;53(5):451-8. 9. Skerfving S, Bergdahl IA. Lead. In: Nordberg G, Fowler B, Nordberg M, Friberg L, editors. Handbook on the Toxicology of Metals. 3rd ed. NY: Academic Press; 2007. p. 599-643. 10. Sommar JN, Svensson MK, Björ BM, Elmståhl SI, Hallmans G, Lundh T, et al. End-stage renal disease

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Еpidemiological Data on Lead Tissue Concentration in Game Birds Induced by Lead Pellets

References Bellrose, F. C., 1959. Lead poisoning as a mortality factor in waterfowl populations. Bulletin of the Illinois Natural History Survey, 27, 231-288. Buerger, T. T., R. E. Mirarchi, M. E. Lisano, 1986. Effects of lead shot ingestion on captive mourning dove survivability and reproduction. Journal of Wildlife Management, 50, 1-8. Butler, D. A., R. B. Sage, R. A. H. Draycott, J. P. Carroll, D. Potts. 2005. Lead exposure in ring-necked pheasants on shooting estates in Great Britain, Wildlife Society

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In vivo quercetol effect in lead acetate poisoning


The present study investigated the influence of quercetol upon δ-aminolevulinic acid (△ -ALA) urine concentration as marker of lead poisoning. The study was conducted on six lots of 6 mature Wistar rats of both sexes, lots not poisoned treated with different concentrations of quercetol (Q1, Q2), control (L6M), lot poisoned untreated (L3Pb), lots poisoned and treated with Q (L4Q1Pb and L5Q2Pb). After 11 days urine from 24 hours was collected for △-ALA spectrophotometric assay and testing the significance of mean difference of by "t" test Student at p <0.05. Statistical analysis of the data presented shows that compared to L2Q2 and L6M the amount of δ-ALA excreted in urine under quercetol influence (L4Q1Pb) shows statistical significance compared to (L2Q2) the amount of δ-ALA excreted in urine compared to (L3Pb) shows statistical significance. Different concentrations of quercetol (Q1, Q2), did not produce significant changes in the δ-ALA excreted compared with values of (L3Pb). Difference between means is probably due to sampling fluctuation, is not significant, reduced growth to eliminate δ-ALA on L4Q1Pb and L5Q2Pb is believed to be due to iron complex formation, reducing hemoglobin synthesis. From the results we conclude that hem biosynthesis does not start to grow under quercetol protection. The obtained data are not relevant statistical since interpretations were performed on non homogeneous groups in number of individuals, the percentage of mortality variability and high levels of standard deviation calculated from each lot.

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Oxidative Stress in Painters Exposed to Low Lead Levels

:1196-202. Gurer-Orhan H, Sabir HU, Ozgunes H. Correlation between clinical indicator of lead poisoning and oxidative stress parameters in controls and lead exposed workers. Toxicology 2004; 195:147-54. Kasperczyk S, Birkner E, Kasperczyk A, Kasperczyk J. Lipids, lipid peroxidation and 7-ketocholesterol in workers exposed to lead. Hum Exp Toxicol 2005;24:287-95. Muntener P, He J, Vupputuri S, Coresh J, Batuman V. Blood lead and chronic kidney disease in the general United States population: results from NHANES III. Kidney

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A Comparative Assessment of Serum Vitamin C and Serum Lead Among Periodontitis and Diabetic Patient

Original Study. RJPBCS 3: 934-939, 2012. 15. Sies H, Jones DP . Oxidative stress. Encyclopedia of stress 3: 45-58, 2007. 16. Taylor RJ . Fact sheet: Nutrients that reduce lead poisoning. 2010. Accessed at: 17. Saraiva MCP, Taichman RS, Braun T, Nriagu J, Eklund SA, Burt BA . Lead exposure and periodontitis in US adults. J Periodont Res 42: 45-52, 2007. 18. Meerwaldt R, Links T, Zeebregts C, Tio R, Hillebrands JL, Smit A. The clinical relevance of

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Clinical, Haematological, and Neurocognitive Findings in Lead-Exposed Workers of a Battery Plant in Iran

References 1. Patrick L. Lead toxicity, a review of the literature. Part 1: Exposure, evaluation, and treatment. Altern Med Rev 2006;11:2-22. PMID: 16597190 2. Staudinger KC, Roth VS. Occupational lead poisoning. Am Fam Physician 1998;57:719-26. 3. Karrari P, Mehrpour O, Abdollahi M. A systematic review on status of lead pollution and toxicity in Iran; Guidance for preventive measures. Daru 2012;20:2. doi: 10.1186/1560-8115-20-2 4. Abdollahi M, Sadeghi Mojarad A, Jalali N. Lead toxicity

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