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A high-MUFA diet alone does not affect ketone body metabolism, but reduces glycated hemoglobin when combined with exercise training in diabetic rats

blood glucose levels [ 3 - 5 ], increased insulin sensitivity, increased glucose homeostasis, lowered FFA, and decreased triglyceride synthesis from the liver [ 4 - 7 ]. Furthermore, exercise training is reported to reduce plasma FFA and β-hydroxybutyric acid levels in a rat model of diabetes induced by streptozotocin (STZ) [ 8 - 10 ], through a reversal of defective activity of 3-ketoacyl-CoA transferase in muscle ketone use [ 9 ], and a decrease in the overall activity of ketone synthesis pathway in the liver [ 10 ]. Monounsaturated fatty acids (MUFA), which are

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