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Introduction Liver cir rhosis (LC) is a common gastroenterological pathology among adults. Its aetiological factors are: alcohol abuse, hepatitis B infection, hepatitis C infection, non-alcoholic fatty liver disease, non-alcoholic steatohepatitis and others.[ 1 , 2 ] Cirrhosis with multiple-aetiologies are more susceptible of developing multiple organ failures, predominantly kidney, brain, heart and others.[ 1 , 3 ] This disease can be classified using several methods and these cases will be focusing on Halstead-Reitan and Child-Pugh score classification

. 4. Kim G, Kang SH, Kim MY, Baik SK. Prognostic value of sarcopenia in patients with liver cirrhosis: a systematic review and meta-analysis. PLoS One. 2017; 12:e0186990. . 5. Montano-Loza AJ, Duarte-Rojo A, Meza-Junco J et al. Inclusion of sarcopenia within MELD (MELDSarcopenia) and the prediction of mortality in patients with cirrhosis. Clin Transl Gastroenterol. 2015; 6:e102. 6. Bhanji RA, Moctezuma-Velazquez C, Duarte-Rojo A et al. Myosteatosis and sarcopenia are associated with hepatic encephalopathy in patients

. The detection of bacterial DNA in blood of rats with CCl4-induced cirrhosis with ascites represents episodes of bacterial translocation. Hepatology 2006;44:633-639. 8. Wiest R, Garcia-Tsao G. Bacterial translocation (BT) in cirrhosis. Hepatology 2005;41:422-433. 9. Sánchez E, Casafont F, Guerra A, de Benito I, Pons-Romero F. Role of intestinal bacterial overgrowth and intestinal motility in bacterial translocation in experimental cirrhosis.Rev Esp Enferm Dig 2005;97(11):805-814. 10. Bauer TM, Schwacha H, Steinbrückner B, Brinkmann FE, Ditzen AK, Aponte JJ, et al

References 1. EUROPEAN ASSOCIATION FOR THE STUDY OF THE LIVER. EASL clinical practice guidelines on the management of ascites, spontaneous bacterial peritonitis and hepatorenal syndrome in cirrhosis. J Hepatol. 2010; 53(3):397-417. 2. SCHRIER RW. Water and sodium retention in edematous disorders: Role of vasopressin and aldosterone. Am J Med. 2006; 119(7 Suppl 1):S47-53. 3. ADROGUÉ HJ, MADIAS NE. Hyponatremia. N Engl J Med. 2000; 342(21):1581-9. 4. BIGGINS SW, RODRIGUEZ HJ, BACCHETTI P, BASS NM, ROBERTS JP, TERRAULT NA. Serum sodium predicts mortality in patients

Bibliography 1. C. L. Bowlus, M.E.Gershwin – The Diagnosis of Primary Biliary Cirrhosis, Autoimmunity Reviews, Volume 13, Issues 4 – 5, April – May 2014, pg.441 - 444. 2. S. C. Hauser, D. S. Pardi, J. J. Poterucha – Mayo Clinic Gastroenterology and Hepatology Board Review, Third Edition, Mayo Clinic Scientific Press And Informa Healthcare USA, Inc, 2008, Rochester, MN, pg.377-381 3. K. D. Lindor,M. E. Gershwin, R. Poupon, M. Kaplan, N. V. Bergasa, and E. J. Heathcote – Primary biliary cirrhosis, AASLD Hepatology, AASLD Practice Guidelines, Volume 50, Issue 1

Introduction Liver cirrhosis (LC) is characterized by hemodynamic alterations that are displayed through hypertension and hyperdynamic circulation. [ 1 ] Hence, portal hypertension (PH) leads to the development of portosystemic collateral channels and abdominal wall collateral veins. PH is the result of the augmentation of intrahepatic resistance against portal blood flow because of hepatic architectural changes, accounting for the development of regenerative nodules surrounded by fibrotic septa, which are the histological peculiarities of hepatic cirrhosis

;30(1):64-68. 7 Wanless IR. Nodular regenerative hyperplasia, dysplasia, and hepatocellular carcinoma .Am J Gastroenterol 1996;91(5):836-837. 8 McDonald JA, Painter DM, Gallagher ND, McCaughan GW. Nodular regenerative hyperplasia mimicking cirrhosis of the liver. Gut 1990;31(6):725-727. 9 Clouet M, Boulay I, Boudiaf M, Soyer P, Nemeth J, Kiselman R, et al.Imaging features of nodular regenerathyperplasia of the liver mimicking hepatic metastases. Imaging 1999;24(3):258-261.

References 1. Schepis F, Camma C, Niceforo D, Magnano A, Pallio S, Cinquegrani M, et al. Which patients with cirrhosis should undergo endoscopic screening for esophageal varices detection? Hepatology 2001;33:333-8. 2. D’Amico G, Luca A. Natural history. Clinical-hemodynamic correlations. Prediction of the risk of bleeding. Bailliere’s Clin Gastroenterol 1997;11:243-56. 3. Giannini E, Botta F, Borro P, Risso D, Romagnoli P, Fasoli A, et al. Platelet count/spleen diameter ratio: Proposal and validation of noninvasive parameter to predict the presence of oesophageal

-720. 7 He W, He Q. Hepatic perfusion parameters in cirrhosis: dynamic CT measurements correlated with portal vein CT angiography. In the Radiological Society of North America: RSNA 2003 Scientific Papers, 2003, Q08-1267. 8 Weidakmm C, Cejna M, Kramer L, et al . Effects of TIPs on liver perfusion measured by dynamic CT. AJR Am J oentgenol, 2005, 184(2): 505-510. 9 Shintaku K, Nakashige A, Ono C, et al . Appearance of the hepatic artery in multidetector-row CT using a test bolus injection. Nippon Igaku Hoshasen Gakkai Zaashi, 2001, 61(3): 100-102. 10 Miles KA, Hayball

August 01]. 3. Ertle J, Dechêne A, Sowa JP, Penndorf V, Herzer K, Kaiser G, et al. Non-alcoholic fatty liver disease progresses to hepatocellular carcinoma in the absence of apparent cirrhosis. Int J Cancer 2011;128:2436-43. 4. Canbay A, Gieseler RK, Gores GJ, Gerken G. The relationship between apoptosis and non-alcoholic fatty liver disease: An evolutionary cornerstone turned pathogenic. Z Gastroenterol 2005;43:211-7. 5. Eade MN, Cooke WT, Williams JA. Liver disease in Crohn’s disease. A study of 100 consecutive patients. Scand J Gastroenterol 1971;6:199-204. 6. Vajro