George Deenichin, Atanas Kristev, Vesselin Mollov and Valentin Turiiski
Sugrue M, Buhkari Y. Intra-abdominalpressure and abdominal compartment syndrome in acute general surgery. World J Surg 2009;33(6):1123-7.
Sukhotnik I, Mogilner J, Hayari L, et al. Effect of elevated intra-abdominalpressure and 100% oxygen in superior mesenteric artery blood flow and enterocyte turnover in a rat. Pediatric Surg International 2009;24(12):1347-53.
Cheatham ML. Nonoperative management of intra-abdominal hypertension and abdominal compartment syndrome. World
Aleksandra Gavrilovska-Brzanov, Zorka Nikolova, Nikola Jankulovski, Mirjana Sosolceva, Gordana Taleska, Maja Mojsova-Mijovska, Marija Jovanovski-Srceva, Darko Angusev, Darko Sazdov and Nikola Brzanov
of abdominalpressure increases pulmonary edema in oleic acid-induced lung injury. Am J Respir Crit Care Med. 2004; 169:534-541.
10. Hunter JD, Damasi Z. Intraabdominal hypertension and the abdominal compartment syndrome. Anaesthesia. 2004; 59:899.
11. Sieh KM, Chu KM, Wong J. Inrtaabdominal hypertension and the abdominal compartment syndrome. Langenbeck's Arch Surg. 2001; 386:53.
12. Moore FK, Hargest R, Martin M, Delicata RJ. Intraabdominal hypertension and the abdominal compartment syndrome. Br. J. Surg. 2004; 91
Gabriel Alexandru Popescu, Tivadar Bara and Paul Rad
ACS: Abdominal Compartment Syndrome
IAH: Intra-Abdominal Hypertension
WSACS: World Society of Abdominal Compartment Syndrome
DL: Decompressive Laparotomy
OA: Open Abdomen
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Raina G. Ardasheva, Mariana D. Argirova, Valentin I. Turiiski and Athanas D. Krustev
1. Holodinsky JK, Roberts DJ, Ball CG, et al. Risk factors for intra-abdominal hypertension and abdominal compartment syndrome among adult intensive care unit patients: a systematic review and meta-analysis. Crit Care 2013;17(5):R249.
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Claudiu Puiac, Theodora Benedek, Lucian Puscasiu, Nora Rat, Emoke Almasy and Janos Szederjesi
. J Crit Care Med . 2016;2:80-84.
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Hektor Sula, Rudin Domi, Arben Beqiri and Andi Koraqi
and Abdominal Compartment Syndrome. II. Recommendations. Intensive Care Med. 2007;33:951-962.
Papavramidis TS, Duros V, Michalopoulos A, Papadopoulos VN, Paramythiotis D, Harlaftis N. Intra-abdominalpressure alterations after large pancreatic pseudocyst transcutaneous drainage. BMC Gastroenterol. 2009;9:42.
Papavramidis TS, Zandes N, Hatzimisios K, Koutsimani Th, Kehagia F, Agorastou P, Doulgerakis M, Patoulidis I. Acute gallstone pancreatitis: a constant challenge for the surgeon. Indian J Surg. 2008
Claudiu Puiac, Janos Szederjesi, Alexandra Lazăr, Codruța Bad and Lucian Pușcașiu
Introduction: Elevated intraabdominal pressure (IAP) it is known to have an impact on renal function trough the pressure transmitted from the abdominal cavity to the vasculature responsible for the renal blood flow. Intraabdominal pressure is found to be frequent in intensive care patients and also to be a predictor of mortality. Intra-abdominal high pressure is an entity that can have serious impact on intensive care admitted patients, studies concluding that if this condition progresses to abdominal compartment syndrome mortality is as high as 80%.
Aim: The aim of this study was to observe if a link between increased intraabdominal pressure and modification in renal function exists (NGAL, creatinine clearance).
Material and Method: The study enrolled 30 critically ill patients admitted in the Intensive Care Unit of SCJU Tîrgu Mures between November 2015 and August 2016. The study enrolled adult, hemodynamically stable patients admitted in intensive critical care - defined by a normal blood pressure maintained without any vasopressor or inotropic support, invasive monitoring using PICCO device and abdominal pressure monitoring.
Results: The patients were divided into two groups based on the intraabdominal pressure values: normal intraabdominal pressure group= 52 values and increased intraabdominal group= 35 values. We compared the groups in the light of NGAL values, 24 hours diuresis, GFR and creatinine clearance. The groups are significantly different when compared in the light of NGAL values and GFR values. We obtained a statistically significant correlation between NGAL value and 24 hour diuresis. No other significant correlations were encountered between the studied items.
Conclusions: NGAL values are increased in patients with high intraabdominal pressure which may suggest its utility as a cut off marker for patients with increased intraabdominal pressure. There is a significant decreased GFR in patient with elevated intraabdominal pressure, observation which can help in early detection of renal injury in patients due to high intraabdominal pressure. No correlation was found between creatinine clearance and increased intraabdominal pressure.
Valentin I. Turiyski, Petar G. Vassilev, Raina G. Ardasheva, Hristo P. Dobrev and Athanas D. Kristev
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Claudiu Puiac, Janos Szederjesi, Alexandra Lazar, Emoke Almasy, Paul Rad and Lucian Puscasiu
secondary abdominal compartment syndrome. Burns. 2014;40:120–6.
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Ramazan Yildiz, Mahmut Ok, Merve Ider, Ugur Aydogdu, Amir Naseri, Kurtulus Parlak and Erdem Gulersoy
as atresia jejuni, coli, recti, or ani, result in the inability of the calves to defecate after birth ( 4 ) and require surgical correction. Intestinal obstruction, which causes pathophysiological changes due to the rise of intra-abdominalpressure, is called abdominal compartment syndrome (ACS). Increasing intra-abdominalpressure causes progressive hypoperfusion and ischaemia of the intestines and other peritoneal and retroperitoneal structures ( 6 ). In newborn infants, ACS usually develops due to congenital gastrointestinal atresia, abdominal wall defects, and