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Effect of Elevated Intra-Abdominal Pressure on the Contractile Activity and Reactivity of Smooth Muscle Tissue from Rat Gastrointestinal Tract to Galantamine and Drotaverine (No-SPA)

References Sugrue M, Buhkari Y. Intra-abdominal pressure and abdominal compartment syndrome in acute general surgery. World J Surg 2009;33(6):1123-7. Sukhotnik I, Mogilner J, Hayari L, et al. Effect of elevated intra-abdominal pressure and 100% oxygen in superior mesenteric artery blood flow and enterocyte turnover in a rat. Pediatric Surg International 2009;24(12):1347-53. Cheatham ML. Nonoperative management of intra-abdominal hypertension and abdominal compartment syndrome. World

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Evaluation of the Effects of Elevated Intra-abdominal Pressure on the Respiratory Mechanics in Mechanically Ventilated Patients

of abdominal pressure increases pulmonary edema in oleic acid-induced lung injury. Am J Respir Crit Care Med. 2004; 169:534-541. 10. Hunter JD, Damasi Z. Intraabdominal hypertension and the abdominal compartment syndrome. Anaesthesia. 2004; 59:899. 11. Sieh KM, Chu KM, Wong J. Inrtaabdominal hypertension and the abdominal compartment syndrome. Langenbeck's Arch Surg. 2001; 386:53. 12. Moore FK, Hargest R, Martin M, Delicata RJ. Intraabdominal hypertension and the abdominal compartment syndrome. Br. J. Surg. 2004; 91

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Abdominal Compartment Syndrome as a Multidisciplinary Challenge. A Literature Review

Abbreviations ACS: Abdominal Compartment Syndrome IAP: Intra-Abdominal Pressure IAH: Intra-Abdominal Hypertension WSACS: World Society of Abdominal Compartment Syndrome DL: Decompressive Laparotomy OA: Open Abdomen R eferences 1. Malbrain ML. You don’t have any excuse, just start measuring abdominal pressure and act upon it. Minerva Anestesiol. 2008;74:1-2. 2. Marginean CO, Melit LE, Chincesan M, et al. Communication skills in pediatrics – the relationship between pediatrician and child. Medicine. 2017;96(43):e8399

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Biochemical Changes in Experimental Rat Model of Abdominal Compartment Syndrome

References 1. Holodinsky JK, Roberts DJ, Ball CG, et al. Risk factors for intra-abdominal hypertension and abdominal compartment syndrome among adult intensive care unit patients: a systematic review and meta-analysis. Crit Care 2013;17(5):R249. 2. Aydin HR, Kesici S, Kesici U, et al. Effects of different intra-abdominal pressure values on different organs: what should be the ideal pressure? Eur Sur 2014;46(5):203-8. 3. Malbrain ML, Cheatham ML, Kirkpatrick A, et al. Results from the international conference of

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Reduction of Intra-abdominal Hypertension Is Associated with Increase of Cardiac Output in Critically Ill Patients Undergoing Mechanical Ventilation

. J Crit Care Med . 2016;2:80-84. 12. Soler Morejon Cde D, Tamargo Barbeito TO. Effect of mechanical ventilation on intra-abdominal pressure in critically ill patients without other risk factors for abdominal hypertension: an observational multicenter epidemiological study. Ann Intensive Care . 2012;2:S22. 13. Rafiei MR, Aghadavoudi O, Shekarchi B, Sajjadi SS, Masoudifar M. Can selection of mechanical ventilation mode prevent increased intra-abdominal pressure in patients admitted to the intensive care unit?. Int J Prev Med . 2013;4:552-556. 14

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Abdominal Compartment Syndrome as an Independent Mortality Predictor Factor During Acute Pancreatitis

and Abdominal Compartment Syndrome. II. Recommendations. Intensive Care Med. 2007;33:951-962. Papavramidis TS, Duros V, Michalopoulos A, Papadopoulos VN, Paramythiotis D, Harlaftis N. Intra-abdominal pressure alterations after large pancreatic pseudocyst transcutaneous drainage. BMC Gastroenterol. 2009;9:42. Papavramidis TS, Zandes N, Hatzimisios K, Koutsimani Th, Kehagia F, Agorastou P, Doulgerakis M, Patoulidis I. Acute gallstone pancreatitis: a constant challenge for the surgeon. Indian J Surg. 2008

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Neutrophil Gelatinase-Associated Lipocalin as a Marker for Renal Dysfunction Detection in Critically Ill Patients with Increased Intraabdominal Pressure


Introduction: Elevated intraabdominal pressure (IAP) it is known to have an impact on renal function trough the pressure transmitted from the abdominal cavity to the vasculature responsible for the renal blood flow. Intraabdominal pressure is found to be frequent in intensive care patients and also to be a predictor of mortality. Intra-abdominal high pressure is an entity that can have serious impact on intensive care admitted patients, studies concluding that if this condition progresses to abdominal compartment syndrome mortality is as high as 80%.

Aim: The aim of this study was to observe if a link between increased intraabdominal pressure and modification in renal function exists (NGAL, creatinine clearance).

Material and Method: The study enrolled 30 critically ill patients admitted in the Intensive Care Unit of SCJU Tîrgu Mures between November 2015 and August 2016. The study enrolled adult, hemodynamically stable patients admitted in intensive critical care - defined by a normal blood pressure maintained without any vasopressor or inotropic support, invasive monitoring using PICCO device and abdominal pressure monitoring.

Results: The patients were divided into two groups based on the intraabdominal pressure values: normal intraabdominal pressure group= 52 values and increased intraabdominal group= 35 values. We compared the groups in the light of NGAL values, 24 hours diuresis, GFR and creatinine clearance. The groups are significantly different when compared in the light of NGAL values and GFR values. We obtained a statistically significant correlation between NGAL value and 24 hour diuresis. No other significant correlations were encountered between the studied items.

Conclusions: NGAL values are increased in patients with high intraabdominal pressure which may suggest its utility as a cut off marker for patients with increased intraabdominal pressure. There is a significant decreased GFR in patient with elevated intraabdominal pressure, observation which can help in early detection of renal injury in patients due to high intraabdominal pressure. No correlation was found between creatinine clearance and increased intraabdominal pressure.

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Study on in Vivo Pial Vessels Alterations and Activity of Isolated Vascular Smooth Muscles in Abdominal Hypertension Rats

REFERENCES 1. Sanchez NC, Tenofsky PL, Dort JM, et al. What is normal intra-abdominal pressure? The American Surgeon 2001;67(3):243-8. 2. Hunt L, Frost SA, Hillman K, et al. Management of intra-abdominal hypertension and abdominal compartment syndrome: a review. J Trauma Manag Outcomes 2014;8:2. 3. Rogers WK, Garcia L. Intra-abdominal hypertension, abdominal compartment syndrome, and the open abdomen. Chest 2018;153(1):238-50. 4. Berry N, Fletcher S. Abdominal compartment syndrome. Continuing Education in Anaesthesia Critical Care & Pain

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Influence of Ventilation Parameters on Intraabdominal Pressure

secondary abdominal compartment syndrome. Burns. 2014;40:120–6. 13. Obeid F, Saba A, Fath J, et al. Increases in intra-abdominal pressure affect pulmonary compliance. Arch Surg. 1995;130:544-7. 14. Krebs J, Pelosi P, Tsagogiorgas C, et al. Effects of positive endexpiratory pressure on respiratory function and hemodynamics in patients with acute respiratory failure with and without intra-abdominal hypertension: a pilot study. Crit Care. 2009;13:R160. 15. Ferrón R, Pedregosa AT, García R, A, et al. Intraabdominal and Thoracic Pressures in Critically Ill

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Evaluation of intestinal damage biomarkers in calves with atresia coli

as atresia jejuni, coli, recti, or ani, result in the inability of the calves to defecate after birth ( 4 ) and require surgical correction. Intestinal obstruction, which causes pathophysiological changes due to the rise of intra-abdominal pressure, is called abdominal compartment syndrome (ACS). Increasing intra-abdominal pressure causes progressive hypoperfusion and ischaemia of the intestines and other peritoneal and retroperitoneal structures ( 6 ). In newborn infants, ACS usually develops due to congenital gastrointestinal atresia, abdominal wall defects, and

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