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References 1. Romagnani S. Human Th17 cells. Arthritis Research and Therapy 2008;10:206-2013. 2. Romagnani S, Maggi E, Liotta F, Cosmi L, Annunziato F. Properties and origin of human Th17 cells. Molecular Immunology 2009;47:3-7. 3. Zhu J, Yamane H, Paul WE. Differentiation of effector CD4 T cell populations. Annu Rev Immunol 2010;28:445-489. 4. Cosmi L, Maggi L, Santarlasci V, Liotta F, Annunziato F. T helper cells plasticity in inflammation. Cytometry Part A 2014;85A;36-42. 5. Annunziato F, Cosmi L, Santarlasci V, Maggi L, Liotta F, Mazzinghi B, Parente E, Fili

) virus. JAMA 2009;301:1042-1046. 19. Wei L, Laurence A, Elias KM, O’Shea JJ. IL-21 is produced by Th17 cells and drives IL-17 production in a STAT3-dependent manner. J Biol Chem 2007;282: 34605-34610. 20. Bettelli E, Carrier Y, Gao W, Korn T, Strom TB, Oukka M, et al. Reciprocal developmental pathways for the generation of pathogenic effector Th17 and regulatory T cells. Nature 2006;441:235-238 21. Veldhoen M, Hocking RJ, Atkins CJ, Locksley RM, Stochinger B. TGFβ in the context of an inflammatory cytokine milieu supports de novo differentiation of IL-17-producing T

References 1. Cakina S, Ocak O, Ozkan A, Yucel S, Karaman HIO. Vitamin D receptor gene polymorphisms in multiple sclerosis disease: A case-control study. Rev Romana Med Lab. 2018;26(4):489-95. DOI: 10.2478/rrlm-2018-0028 2. Arellano G, Acu-a E, Reyes LI, Ottum PA, De Sarno P, Villarroel L, et al. Th1 and Th17 Cells and Associated Cytokines Discriminate among Clinically Isolated Syndrome and Multiple Sclerosis Phenotypes. Front Immunol. 2017 Jun 30; 8:753. DOI: 10.3389/fimmu.2017.00753 3. Harrington LE, Hatton RD, Mangan PR, Turner H, Murphy TL, Murphy KM, et al

CRSsNP; however, we didn’t find any impact of Th1 cell count on disease control and speculated that Th1 cells were not important for the pathogenesis in CRSwNP. 13 CRSsNP was found to have more heterogeneous endotypes, with either Th1, Th2 or Th17 cytokines; furthermore, the remodelling process was not in correlation with CRSsNP endotype. 4 , 9 , 14 Th17 cells can secrete IL-17, IL-22 or IFN-γ upon stimulation. 15 Moreover, in our recent flow cytometric study, we found that Th17 cells in CRSwNP were associated with well-controlled CRSwNP. 13 Similarly, Th17 were

REFERENCES 1. GRAEBER KE, OLSEN NJ. Th17 cell cytokine secretion profile in host defense and autoimmunity . Inflamm Res. 2012; 61 (2):87-96. 2. HYMOWITZ SG, FILVAROFF EH, YIN JP, LEE J, CAI L, RISSER P, et al. IL-17s adopt a cystine knot fold: structure and activity of a novel cytokine, IL-17F, and implications for receptor binding . EMBO J. 2001; 20 :5332–41. 3. TANASESCU C, BĂLĂNESCU E, BĂLĂNESCU P, OLTEANU R, BADEA C, GRANCEA C, et al. IL-17 in cutaneous lupus erythematosus . Eur J Intern Med. 2010; 21 (3):202-7. 4. BLAUVELT A. T-helper 17 cells in

References 1. Hollenbach JA, Thompson SD, Bugawan TL, et al. Juvenile idiopathic arthritis and HLA class I and class II interactions and age-at-onset effects. Arthrit Rheumat. 2010;62:1781-91. 2. Reiner SL. Development in motion: Helper T cells at work. cell. 2007;129(1):33-6. 3. Tesmer LA, Lundy SK, Sarkar S, Fox DA. Th17 cells in human disease. Immunol Rev. 2008;223:87-113. 4. Ouyang W, Filvaroff E, Hu Y, Grogan J. Novel therapeutic targets along the Th17 pathway. Eur J Immunol. 2009;39(3):634-75. 5. Pappu R, Ramirez-Carrozzi V, Sambandam A. The interleukin-17

. [ 8 ] have observed from in vitro implant bone model that Brucella can infect and replicate in bone marrow-derived dendritic cells (DCs) through granulocyte-macrophage colony-stimulating factor. Gao et al. [ 9 ] have discovered that when Brucella invades the host, the function of natural killer (NK) cells is inhibited, which can cause abortion. When type 1 T helper (Th1) cell is downregulated, the in vivo Th1 cell/Th2 cell imbalance can also cause the animal host to abort [ 10 , 11 ]. Th17 cells provide the host organism with additional defense against a number

. Interleukin-17-producing CD4(+) T cells increase with severity of liver damage in patients with chronic hepatitis B. Hepatology 2010;51:81-91. 10 Zhang JY, Song CH, Shi F, Zhang Z, Fu JL, Wang FS. Decreased ratio of Treg cells to Th17 cells correlates with HBV DNA suppression in chronic hepatitis B patients undergoing entecavir treatment. PLoS One 2010;5:e13869. 11 Piao RL, Liu YY, Tian D, Ma ZH, Zhang M, Zhao C, Niu JQ. Adefovir dipivoxil modulates cytokine expression in Th1/Th2 cells in. Mol Med Report 2012;5:184-189. 12 Nischalke HD, Vogel M, Mauss S, Baumgarten A, Lutz

. Psychiatry Res. 2015; 225(1-2): 58-63. 51. Dimitrov DH, Lee S, Yantis J, Valdez C, Paredes RM, Braida N, Velligan D, Walss-Bass C. Differential correlations between inflammatory cytokines and psychopathology in veterans with schizophrenia: potential role for IL-17 pathway. Schizophr Res. 2013; 151 (1-3): 29-35. 52. Ding M, Song X, Zhao J, Gao J, Li X, Yang G, Wang X, Harrington A, Fan X, Lv L. Activation of Th17 cells in drug naïve, first episode schizophrenia. Prog Neuropsychopharmacol Biol Psychiatry.2014; 51: 78-82. 53. Tourjman V, Kouassi É, Koué MÈ, Rocchetti M

Immunol. 2008; 38: 2636-2649. 40. Bunte K, Beikler T. Th17 Cells and the IL-23/IL-17 Axis in the Pathogenesis of Periodontitis and Immune-Mediated Inflammatory Diseases. Int J Mol Sci. 2019; 20. 41. Chehimi M, Vidal H, Eljaafari A. Pathogenic Role of IL-17-Producing Immune Cells in Obesity, and Related Inflammatory Diseases. J Clin Med. 2017; 6. 42. Harrell CR, Simovic Markovic B, Fellabaum C, Arsenijevic A, Djonov V, Arsenijevic N, Volarevic V. Therapeutic Potential of Mesenchymal Stem Cell-Derived Exosomes in the Treatment of Eye Diseases. Adv Exp Med Biol. 2018; 1089