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Zhong-ying Bao, Xiao Ming, Xiao-dong Yuan and Shu-hong Duan

References 1. Huang YL. Clinical Infectious Diseases. Beijing: People’s Medical Publishing House, 1990:246-253. 2. Peng WW. Epidemiology fifth edition. Beijing: People’s Health Publishing House, 2002:156-157. 3. Harris JB, LaRocque RC, Qadri F, Ryan ET, Calderwood SB. Cholera. Lancet 2012;379(9835):2466-2476. 4. Jiang LJ, Wang R, Qiao Y, Wang BR. O1 group O139 and non-O1 Vibrio cholerae virulence comparative study. Adv Microbial Immunol 1995; 23:1-11. 5. Duan YQ, Yang B, Cui JD

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Jin Zhang

.1016/j.molimm.2015.01.009 Golshani M. Rafati S. Dashti A. Gholami E. Siadat S.D. Oloomi M. Vaccination with recombinant L7/L12-truncated Omp31 protein induces protection against Brucella infection in BALB/c mice Mol. Immunol. 2015 65 2 287 292 [2] Tian M., Qu J., Bao Y., Gao J., Liu J., Wang S., et al., Construction of pTM series plasmids for gene expression in Brucella species, J. Microbiol. Methods, 2016, 123, 18-23. Tian M. Qu J. Bao Y. Gao J. Liu J. Wang S. Construction of pTM series plasmids for gene expression in Brucella species J. Microbiol. Methods

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Yuan Liu

References 1 Jiang J, Duo L. Study on the Correlation between Psoriasis Vulgaris and HLADRB1*07 allele in Xinjiang Uygur Autonomous Region. Journal of Medical Postgraduates, 2012, 25 (1): 54-57. 2 Hort W, Mayser P. Malassezia virulence determinants. Curr Opin Infect Dis, 2011, 24(2): 100-105. 3 Rup E, Skora M, Krzysciak P, et al . Distribution of Malassezia species in patients with psoriasis-quality assessment. Postep Dermatol Alergol, 2010, 27(4): 264-268. 4 Gaitanis G, Magiatia P, Hantschke M, et al . The Malassezia genus in skin

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Ming Guo and Jingfeng Zhang

Transfusion Infectivity 2.1 Effects of HBV Genotype According to their entire gene sequences, differences among various HBV strains reach >8%, and to date, 10 genotypes (from A to J) have been discovered. Evidence indicates that different HBV genotypes lead to different HBV infectivities through blood transfusions. Using experiments on gorillas infected with HBV, the American group Hsia et al . [ 4 ] proved that for different genotypes, 50% chimp infectious dose (CID 50 ) of HBV differs. The C genotype spans three genome equivalents (geq, D genotype totals 78 geq

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Lingling Zu

bacilli in the stomach of patients with gastritis and peptic ulceration. Lancet, 1984, 1(8390):1311-1315. 6 Alakkari A, Zullo A, O’Connor HJ. Helicobacter pylori and nonmalignant diseases. Helicobacter, 2011, 16 (Suppl1):33-37. 7 Cover T, Peek RM. Diet, microbial virulence, and Helicobacter pylori- induced gastric cancer. Gut Microbes, 2013,4(6):482-493. 8 Krajden S, Fuksa M, Anderson J, et al . Examination of human stomach biopsies, saliva, and dental plaque for Campylobacterpylori. J Clin Microbiol, 1989, 27(6):1397-1398. 9 Zou QH, Li RQ

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Sara Batoei, Mohaddese Mahboubi and Reza Yari

EZ. Distribution and evolution of virulence factors in septicemic Escherichia coli . Int J Med Microbiol 2010; 300(6):367-370. doi: 11. Arshad M, Seed PC. Urinary tract infections in the infant. Clin Perinatol 2015; 42(1):17-28. doi: 12. Saltoglu N, Karali R, Yemisen M, Ozaras R, Balkan II, Mete B, et al. Comparison of community onset healthcare associated and hospital acquired urinary infections caused by extended spectrum beta lactamase producing Escherichia

Open access

Dan Wang

bacteria and fungi: (1) the physical support of surface structures (such as cell wall, mycelium, flagella, and cilia) [ 21 ] and (2) through interactions of secretion or autolysis chemicals (such as signaling molecules, extracellular polysaccharide, protein, and DNA and RNA) to complete the exchange and information transmission of genes and metabolites, change antibiotic resistance, adapt to environment pressures, and express virulence genes. Given the complexity of the interaction between bacteria and fungi, scarce information can explain the specific mechanisms, which

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E. Villar-Luna, O. Goméz-Rodriguez, R. I. Rojas-Martínez and E. Zavaleta-Mejía

carrying dominant genes conferring resistance to the three major species of RKNs on tomato ( Mi-1 ), potato ( Mh ), soybeans (Mir1) and pepper (N and Tabasco) ( Castagnone-Sereno, 2012 ). Meloidogyne enterolobii has been detected in Africa, Europe, USA, Central and South America ( Brito et al ., 2007 ; Moens et al., 2009 ; Castagnone-Sereno, 2012 ); and recently was first reported in Mexico parasitizing watermelon (Veracruz state) and tomato plants (Sinaloa state) ( Ramirez-Suarez et al ., 2014 ; Martinez et al ., 2015 ); however, its presence in other crops

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Xiaoming Gu

]. As a common clinical conditional pathogen, UPEC is also known as the molecular fimbrial chaperone/usher pathway (CUP) [ 6 ], and 38 different CUP expression regions have been identified in the gene spectrum of E . coli . A single UPEC strain expresses 12 different fimbrial CUPs [ 7 ]. In a mouse model, type I pili was found to be necessary for supporting UPEC colonization, invasion, and maintenance of continuous infection in the bladder [ 8 ]. After type I pili binds with the FmH adhesion, glycated urinary plaque proteins and α1β3 integrins can be recognized in

Open access

Hang Li

, Piqueres P, Alonso JL, et al . Survival and viability of Helicobacter pylori after inoculation into chlorinated drinking water. Water Res, 2007, 41:3490-3496. 33 Samra ZQ, Javaid U, Ghafoor S, et al . PCR assay targeting virulence genes of Helicobacter pylori isolated from drinking water and clinical samples in Lahore metropolitan, Pakistan. J Water Health, 2011, 9: 208-216. 34 Hulten K, Han SW, Enroth H, et al . Helicobacter pylori in the drinking water in Peru. Gastroenterology, 1996, 110:1031-1035. 35 Horiuchi T, Ohkusa T, Watanabe M, et al