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Yuliya I. Ragino, Viktoriya S. Shramko, Ekaterina M. Stakhneva, Elena I. Chernyak, Sergey V. Morozov, Elena V. Shakhtshneider, Yana V. Polonskaya, Liliia V. Shcherbakova and Alexander M. Chernyavskiy

study was conducted within the framework of R&D topics of Government contracts No. 0324-2018-0002 and 0324-2017-0048 and with the financial support of RFBR grant No. 17-04-02120. The study protocol was approved by the local Ethics Committee of the Institute of Internal and Preventive Medicine (a branch of the Institute of Cytology and Genetics, the Siberian Branch of the Russian Academy of Sciences, Novosibirsk, Russia). Each patient gave written informed consent to be examined and to participate in the study. The study was carried out in the two groups comparable

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Zekiye Catak, Esra Kocdemir, Kader Ugur, Meltem Yardim, İbrahim Sahin, Hilal Kaya and Suleyman Aydin

Introduction Schizophrenia is a complex psychotic disorder, and its pathophysiology is not yet clear ( 1 ). Until now, the dopamine (DA) theory, one of the biochemical theories of schizophrenia pathophysiology, has received the most attention ( 2 , 3 ). This theory suggests an overactive state in dopaminergic stimulation in certain brain regions of patients ( 2 , 4 ). The dopamine theory of schizophrenia is the principal basis of antipsychotic drug treatment ( 5 , 6 , 7 ). On the other hand, it has been argued that genes involved in the dopaminergic