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Blesneac Cristina, Şuteu Carmen, Togănel Rodica, Benedek Theodora and I Benedek

Study. Circulation 1995; 92:785–789. 4. Luis E. Alday and Eduardo Moreyra (2012). Hypertrophic Cardiomyopathy in Infants and Children, Cardiomyopathies - From Basic Research to Clinical Management, Prof. Josef Veselka (Ed.), ISBN: 978-953-307-834-2. 5. Lipshultz SE, Sleeper LA, Towbin JA, et al. The incidence of pediatric cardiomyopathy in two regionsof the United States. N Engl J Med 2003;348(17):1647–1655. 6. Colan SD, Lipshultz SE, Lowe AM, et al., Epidemiology and causespecificoutcome of hypertrophic cardiomyopathy in children:Findings from the

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Ina E. Geneva, Maya B. Krasteva and Stefan S. Kostianev

Potentials. In: E-medicine. Updated: Feb 9, 2012. [Internet]. Available from: 5. Geneva IE, Krasteva MB, Kostianev SS. Age-related changes of the somatosensory evoked potential in healthy children. Folia Med (Plovdiv) 2002;44(4):13-18. 6. George SR, Taylor MJ. Somatosensory evoked potentials in neonates and infants: developmental and normative data. Electroenceph Clin Neurophysiol 1991;80:94-102. 7. Taylor MJ, Fagan ER. SEPs to median nerve stimulation: normative data for paediatrics. Electroenceph Clin

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Ivan S. Ivanov, Nikolay I. Popov, Rumyana I. Moshe, Dora D. Terzieva, Rumen S. Stefanov, Margarita V. Panova, Maria V. Atanasova and Ben Zion Garty

-Pinhata MM, Yamamoto AY, Moura Brito RM, et al. Birth prevalence and natural history of congenital cytomegalovirus infection in a highly seroimmune population. Clin Infect Dis 2009;49(4):522-8. 5. Vaudry W, Rosychuk RJ, Lee BE, et al. Congenital cytomegalovirus infection in high-risk Canadian infants: Report of a pilot screening study. Can J Infect Dis Med Microbiol 2010;21(1):e12-9. 6. Karparov A. Cytomegalovirus (CMV). Official Bulletin (Published by Bulgarian Forum for Herpes Infections) 1997;3:9-12 (Bulgarian). 7. Seale H

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David Mage, E. Donner, Mechtild Vennemann, Peter Fleming, Katia Sol-Church, Rebecca Drake and Sam Gulino

References Fleming PJ, Blair PS, Bacon C, Berry PJ. Sudden Unexpected Death In Infancy. The CESDI SUDI Studies 1993-1996. Pub. The Stationery Office, London, 2000. ISBN 0 11 322299 8. Centers for Disease Control and Prevention (CDC). Sudden Unexpected Infant Death (SUID) Initiative. Shapiro-Mendoza CK, Tomashek KM, Anderson RN, Wingo J. Recent national trends in sudden, unexpected infant deaths: more evidence supporting a change in classification or

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David T. Mage, Maria L. Latorre, Alejandro G. Jenik and E. Maria Donner

REFERENCES [1] Mage DT, Donner M. A genetic basis for the sudden infant death syndrome sex ratio. Med Hypotheses. 1997 Feb;48(2):137-142. [2] Mage DT, Donner EM. The fifty percent male excess of infant respiratory mortality. Acta Paediatr. 2004 Sep;93(9):1210-1215. [3] Mage DT, Donner EM. Is excess male infant mortality from sudden infant death syndrome and other respiratory diseases X-linked? Acta Paediatr. 2014 Feb;103(2):188-193. [4] Mage DT. A probability model for the age distribution of SIDS. JSIDS & Infant Mortality 1996;1 (1

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Vesna D. Živković, Ivona Stanković, Lidija Dimitrijević, Hristina Čolović, Marija Spalević and Nataša Savić

References 1. Kljajić D, Trgovčević S, Stanković M. Birth lesions of plexus brachialis - secondary motor deficit. Health Care 2011;40:69-74. 2. Zafeiriou D, Psychogiou K. Pathogenesis of obstetric brachial plexus palsy. Pediatric Neurol 2008; 38:235-42. 3. Duff SV, DeMatteo C. Clinical assessment of the infant and child following perinatal brachial plexus injury. J Hand Ther 2015;28:126-33. 4. Rota

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Florina-Mădălina Oniceanu, Ileana Ion, Elena Leu, Adriana Apostol, Cecilia Adumitresi, Ninela Rădulescu, Cristina Farcaş, Anabella-Cristiana Ştefănescu and R. Chirică

N, et al. - Retroviral-mediated gene transfer of gp91phox into bone marrow cells rescues defect in host defense against Aspergillus fumigatus in murine X-linked chronic granulomatous disease. Blood. Jan 1 1997;89(1):41-8. [Medline]. 5. BARTON LL, MOUSSA SL, VILLAR RG, HULETT RL. - Gastrointestinal complications of chronic granulomatous disease: case report and literature review. Clin Pediatr (Phila). Apr 1998;37(4):231-6. [Medline]. 6. JESSICA S. KOSUT, MD; NAYNESH R. KAMANI, MD; BARBARA A. JANTAUSCH,MD. - One-Month-Old Infant With

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Slavica Sunarić, Tatjana Jovanović, Ana Spasić, Marko Denić and Gordana Kocić

References 1. World Health Organization. Infant and young child feeding: Model chapter for textbooks for medical students and allied heath professionals. Washington, DC: Author, 2009. 2. Kocic GM, Jevtovic-Stoimenov T, Sokolovic D et al. Milk consumption and chronic disease risk-the strategy or challenge to avoid and eliminate “unwanted” compounds and contaminants. J AgrSci 2015; 7(5): 154-62. 3. Kocic G, BjelakovicLj, Cvetkovic T et al. Enzyme activity of human milk during the first month of lactation

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Thitima Ngoenmak, Julintorn Somran, Chutima Phuaksaman and Jaruwat Khunrat

Thai male infant. He developed jaundice, hepatosplenomegaly, and lethargy within a few days after breast-feeding, and these features persisted until he was 4 months old. Clinical and laboratory improvement confirmed a good response to lactosefree diet. This retrospective study was approved by the Ethics and Institutional Review Committee and Board of Naresuan University. Case report A 4-month-old, Thai male infant was normally delivered at full term (40 weeks of gestational age) as a first son to nonconsanguineous mother. His family has no history of known

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Natasa Ristic, Vladimir Ajdzanovic, Svetlana Trifunovic, Nasta Tanic, Nada Bujisic and Verica Milosevic


The effects of estradiol-dipropionate (EDP) or human chorionic gonadotropin (hCG) on immunohistomorphometric characteristics of pituitary GH cells in infant and peripubertal female rats were investigated. The first group of females received five injections of EDP (0.25 mg/kg b.w.) during the neonatal period of life, and was further divided into two subgroups which were sacrificed at the infantile period (17th day) or at the peripubertal period (38th day). The second group received two doses of hCG (50 IU/kg b.w.) on the 15th and 16th day of life in the first subgroup, and on the 36th and 37th days of life in the second subgroup, while they were sacrificed 24 h after the last treatment, respectively. The control females were injected with an equivalent volume of the vehicle and sacrificed according to the appropriate schedules as the hormone treated rats. EDP treatment decreased GH cell volume density in infant and peripubertal females, by 38% and 76% (p<0.05) respectively, in comparison with the controls. The number of GH cells per mm2 in infantile and peripubertal period was decreased in EDP treated animals by 26% and 53% (p<0.05) respectively, compared to the controls. Also, upon EDP treatment in both periods, GH cells were diminished in size and less intensely immunolabelled than in the control groups. The morphometric parameters in animals treated with hCG were insignificantly changed in both analyzed periods, in comparison with the controls. Unlike hCG, EDP manifested clear inhibitory effects on the immunohistomorphometric characteristics of GH cells in examined female rats.