have been reported.
An investigation of whether AM can attenuate renal fibrosis and aid in processes of protection against and reversal of tissue damage, and the mechanisms of AM action in any such attenuation is warranted. To determine whether AM can disrupt progression of renal fibrosis in a rat model of the disease, histopathology using Sirius Red collagen fiber staining, which allows us to determine the relative amount of collagen fibers in renal tissue, and proteomic analysis of protein marker expression was used to understand better potential mechanisms of AM
artery; RCA, right coronary artery
( A ) One site of the myocardial bridge on the AIA. ( B ) Two sites on the AIA. AIA, anterior interventricular artery
Identification of the dominant pattern of coronary arteries has clinical importance particularly from the functional impact of myocardial ischemia [ 3 ]. The prevalence of right dominance had been reported to be 40%–80% [ 3 , 6 , 7 , 8 ], left dominance 6.8%–18.57% [ 7 , 9 , 10 ], and codominance 10%–42.5% [ 8 , 11 , 12 ]. In the present study, the prevalence of right
Yi-sheng Zhong, Min-hong Xiang, Wen Ye, Ping Huang, Yu Cheng and You-qin Jiang
A, Centofanti M, Mattei E, De Gregorio A, Aloe L, et al. Histopathological evaluation of retinal damage during intraocular hypertension in rabbit: involvement of ganglion cells and nerve fiber layer. Graefes Arch Clin Exp Ophthalmol. 1996; 234 (Suppl 1):S209-13.
16. Zhu MD, Cai FY. Development of experimental chronic intraocular hypertension in the rabbit. Aust N Z Ophthalmol. 1992; 20:225-34.
17. Heijl A, Leske MC, Bengtsson B, Hyman L, Bengtsson B, Hussein M, et al. Reduction of intraocular pressure and glaucoma progression
Onanong Jitkritsadakul, Priya Jagota and Roongroj Bhidayasiri
, brainstem, and higher brain [ 16 ]. The simplest loop is the peripheral monosynaptic stretch reflex loop where the Ia afferent fibers from the muscle spindle synapse directly with the α motor neurons in the spinal cord, which send axons to the extrafusal muscle fibers [ 16 ]. Theoretically, flexion movements will stretch and cause afferent volley eliciting reflexes in the antagonistic extensors. When an extensor muscle is activated, a similar pattern occurs, causing an afferent volley to the flexors [ 23 ]. These reflex loops are then connected and oscillate over time
Beaulieu J, Drolet G, Chanpagne D. Enkephalin innervation of the paraventricular nucleus of the hypothalamus: Distribution of fibers and origins of input. J Chem Neuroanat 10, 79–92, 1996.
Blake NG, Johnson MR, Eckland DJA, Foster OJF, Lightman SL. Effect of food deprivation and altered thyroid status on the hypothalamic-pituitary-thyroid axis in the rat. J Endocrinol 133, 183–188, 1992.
Ceccatelli S, Eriksson M, Hokfelt T. Distribution and coexistence of corticotropin-releasing factor-, neurotensin-, enkephalin-, cholecystokinin-, galanin- and vasoactive
Dora Zelena, Ophelie Menant, Frederic Andersson and Elodie Chaillou
NS. The periaqueductal gray and its potential role in maternal behavior inhibition in response to predatory threats. Behav Brain Res 209, 226-233, 2010.
Swanson LW, McKellar S. The distribution of oxytocin- and neurophysin-stained fibers in the spinal cord of the rat and monkey. J Comp Neurol 188, 87-106, 1979.
Takasaki A, Hui M, Sasaki M. Is the periaqueductal gray an essential relay center for the micturition reflex pathway in the cat? Brain Res 1317, 108-115, 2010.
Tasker RR. Identification of pain processing