Meng Yue, Zhan-Ying Ma, Meng-Jie Lei, Chu-Yun Cui and Yi Jin
identification of included studies.
3.1 Study selection
In this systematic review, the search in all databases yielded 655 potentially relevant articles. Of those, seven registered clinical trials have been completed, but there were no study results posted. After screening the titles and/or abstract, 30 full-text articles were selected ( Figure 1 ). Twenty-two studies failed to meet the inclusion criteria, and thus, eight studies (seven RCTs and one CCT) were included in the qualitative synthesis 20 , 21 , 22 , 23 , 24 , 25 , 26 , 27 and five 20
have been reported.
An investigation of whether AM can attenuate renal fibrosis and aid in processes of protection against and reversal of tissue damage, and the mechanisms of AM action in any such attenuation is warranted. To determine whether AM can disrupt progression of renal fibrosis in a rat model of the disease, histopathology using Sirius Red collagen fiber staining, which allows us to determine the relative amount of collagen fibers in renal tissue, and proteomic analysis of protein marker expression was used to understand better potential mechanisms of AM
artery; RCA, right coronary artery
( A ) One site of the myocardial bridge on the AIA. ( B ) Two sites on the AIA. AIA, anterior interventricular artery
Identification of the dominant pattern of coronary arteries has clinical importance particularly from the functional impact of myocardial ischemia [ 3 ]. The prevalence of right dominance had been reported to be 40%–80% [ 3 , 6 , 7 , 8 ], left dominance 6.8%–18.57% [ 7 , 9 , 10 ], and codominance 10%–42.5% [ 8 , 11 , 12 ]. In the present study, the prevalence of right
Yi-sheng Zhong, Min-hong Xiang, Wen Ye, Ping Huang, Yu Cheng and You-qin Jiang
A, Centofanti M, Mattei E, De Gregorio A, Aloe L, et al. Histopathological evaluation of retinal damage during intraocular hypertension in rabbit: involvement of ganglion cells and nerve fiber layer. Graefes Arch Clin Exp Ophthalmol. 1996; 234 (Suppl 1):S209-13.
16. Zhu MD, Cai FY. Development of experimental chronic intraocular hypertension in the rabbit. Aust N Z Ophthalmol. 1992; 20:225-34.
17. Heijl A, Leske MC, Bengtsson B, Hyman L, Bengtsson B, Hussein M, et al. Reduction of intraocular pressure and glaucoma progression
Onanong Jitkritsadakul, Priya Jagota and Roongroj Bhidayasiri
, brainstem, and higher brain [ 16 ]. The simplest loop is the peripheral monosynaptic stretch reflex loop where the Ia afferent fibers from the muscle spindle synapse directly with the α motor neurons in the spinal cord, which send axons to the extrafusal muscle fibers [ 16 ]. Theoretically, flexion movements will stretch and cause afferent volley eliciting reflexes in the antagonistic extensors. When an extensor muscle is activated, a similar pattern occurs, causing an afferent volley to the flexors [ 23 ]. These reflex loops are then connected and oscillate over time