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Prognosing a severe course of asthma in children following the study of endothelial function

engage into physical activity. Venous blood (2 ml) was taken before meals and centrifuged at 2000 rpm for 10 min. Serum in the lower portion of the test tube was stored at -30°C until the assessment. The collected blood samples were analyzed to determine the serum level of the soluble vascular molecule of intercellular adhesion-1 (sVCAM-1) by immunoassay assay using human sVCAM-1 platinum ELISA (eBioscience, Bender MedSystems GmbH, Austria, BMS232) and also to determine the concentration of stable nitrogen oxide metabolites (NO 2 + NO 3 ) spectrophotometrically

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Risk for stroke and chronic kidney disease in patients with sleep apnea syndrome and heart failure with different ejection fractions

, Moura L, Pierard LA, Tribouilloy C, Hagendorff A, et al. European Association of Echocardiography recommendations for the assessment of valvular regurgitation. Part 1: aortic and pulmonary regurgitation (native valve disease), European Journal of Echocardiography 2010, 11:223–244; Part 2: mitral and tricuspid regurgitation (native valve disease), European Journal of Echocardiography 2010;11:307–332. 10.1093/ejechocard/jeq030 Lancellotti P Moura L Pierard LA Tribouilloy C Hagendorff A et al European Association of Echocardiography

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Hypersensitivity pneumonitis in a teenager

examination was negative for tuberculosis. BAL from the medium lobe bronchus recovered 100 ml from 120 ml instilated and showed a significant hypercellularity: 57.4 × 10 6 cells (normally, for non-smokers, less than 13 × 10 6 cells). The differential cytology identified a lymphocytic alveolitis: macrophages 15.0% (normally >84%), lymphocytes 74.2% (normally <13%), granulocytes 10.4% (normally <3%), neutrophils 9.8% (normally <3%), eosinophils 0.6% (normally <0.5%) and mastocytes 0.4% (normally <0.5%). It should be mentioned that the increased neutrophils percent is

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Decreased plasma epidermal growth factor (EGF) levels in patients with severe chronic obstructive pulmonary disease

This work was personally funded without any grant. References 1 Quaderi SA, Hurst JR. The unmet global burden of COPD. Global Health, Epidemiology and Genomics 2018;3:e4–e4. 29868229 10.1017/gheg.2018.1 Quaderi SA Hurst JR The unmet global burden of COPD Global Health, Epidemiology and Genomics 2018 3 e4 e4 2 Vestbo J, Hurd SS, Agusti AG, Jones PW, Vogelmeier C, Anzueto A, et al. Global strategy for the diagnosis, management, and prevention of chronic obstructive pulmonary disease: GOLD executive summary. American Journal of Respiratory

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Primary Biliary Cirrhosis and Ankylosing Spondylitis, A Rare Association

.A. Torres – Primary biliary cirrrhosis associated with Ankylosing spondylitis, Journal of Clinical Gastroenterology, 1994, 18(3), pg.263 – 264. 10. J. Zochling, D. van der Heijde, R. Burgos-Vargas, E. Collantes, J. C. Davis Jr, B. Dijkmans, M. Dougados, P. Ge´her, R. D. Inman, M. A. Khan, T. K. Kvien, M. Leirisalo- Repo, I. Olivieri, K. Pavelka, J. Sieper, G. Stucki, R. D. Sturrock, S. van der Linden, D. Wendling, H. Bo¨hm, B. J. van Royen, J. Braun – ASAS/EULAR recommendations for themanagement of ankylosing spondylitis, Annals of the Rheumatic Diseases, 2006

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A New Biomarker in Acute-Decompensated Heart Failure. Preliminary Study

, Tandogan I, Yilmaz MB, Gul I, Zorlu A. CA125 in heart failure: Implications for immunoinflammatory activity. Int J Cardiol. 2011;146(1):99-100. doi:10.1016/j.ijcard.2010.05.077 4. Huang F, Zhang K, Chen J, et al. Elevation of carbohydrate antigen 125 in chronic heart failure may be caused by mechanical extension of mesothelial cells from serous cavity effusion. Clin Biochem. 2013;46(16-17):1694-1700. doi:10.1016/j.clinbiochem.2013.09.008 5. Monteiro S, Franco F, Costa S, et al. Prognostic value of CA125 in advanced heart failure patients. Int J Cardiol. 2010

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Osas and Associated Comorbidities - A Retrospective Study

in a large cohort.” Sleep. 2005; 28(11):1405–1411. 16. McCallW, et al. “Correlates of depressive symptoms in patients with obstructive sleep apnoea.” J Clin Sleep Med. 2006;2:424–6. 17. Virend K. Somers, et al. “Sleep Apnea and Cardiovascular Disease.” Journal of the American College of Cardiology Vol. 52, No. 8, 2008, pp.686-717, by the American Heart Association, Inc, and the American College of Cardiology Foundation, Published by Elsevier Inc., doi:10.1016/j.jacc.2008.05.002. 18. Peppard PE, et al. “Prospective study of the association between

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Metabolic Syndrome and Myocardial Involvement in HIV-Infected Patients

, December 2011, pp 898-903. 4. Carr A. HIV lipodystrophy: risk factors, pathogenesis, diagnosis and management. AIDS. 2003;17:S141S148. 5. Barbaro G. Highly active antiretroviral therapy-associated metabolic syndrome: pathogenesis and cardiovascular risk. Am J Therapeut. 2006; 13: 248-60. 6. Triant VA, Lee H, Hadigan C, Grinspoon SK. Increased acute myocardial infarction rates and cardiovascular risk factors among patients with human immunodeficiency virus disease. J Clin Endocrinol Metab 2007; 92:2506. 7. Carr A, Cooper D. Adverse effects of

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The Therapeutic Means of Obstructive Sleep Apnea Syndrome (OSAS)

. “Treatment of the idiopathic respiratory-distress syndrome with continuous positive airway pressure.” PubMed - NCBI”. N Engl J Med. 1971 Jun 17; 284(24):1333-40. 13. Virend K. Somers, et al. “Sleep Apnea and Cardiovascular Disease.” Journal of the American College of Cardiology Vol. 52, No. 8, 2008, pp.686-717, by the American Heart Association, Inc., and the American College of Cardiology Foundation, Published by Elsevier Inc., doi:10.1016/j.jacc.2008.05.002. 14. Tawfic S Hakim, Enrico M Comporesi. “Expiratory and Inspiratory Positive Airway Pressures in

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