Search Results

1 - 10 of 51 items :

  • Public Health x
Clear All
The cooperation between orthodontists and surgeons in treating facial skeletal deformities

orthognathic surgery. Angle Orthod. 2010;80:361-6. 8. Takada K, Petdachai S, Sakaduda M. Changes in dentofacial morphology in skeletal Class III children treated by a modified maxillary protraction headgear and a chin cup: a longitudinal cephalometric appraisal. Eur J Orthod. 1993;15:211-21. 9. Freeman CS, Mc Namara JA Jr, Bacetti T, et al. Treatment effects of the bionator and high-pull facebow combination followed by fixed appliances in patients with increased vertical demensions Am J Orthod Dentofacial Orthop. 2007

Open access
The influence of the sensitivity of the trapezius muscle on the changes within the bioelectric tensions of the masticatory muscles

. Characteristics and distributions of myofascial trigger points in individuals with chronic tension-type headaches. J Phys Ther Sci. 2019;31:306. 7. Moloney TM, Witney AG. Pressure pain thresholds increase after preconditioning 1 Hz repetitive transcranial magnetic stimulation with transcranial direct current stimulation. PLoS ONE. 2014;9:e92540. 8. Wieczorek A, Loster J, Loster B, et al. Correlation between activity and asymmetry indices and skeletal classes in symptomatic-free young adults. J Stomatol. 2013;66:750-60. 9. Okeson JP, Drobek W. Leczenie

Open access
Clinical manifestations of neurofibromatosis type 1 – a diagnostic and management challenge

Abstract

Introduction. Neurofibromatosis type 1 (NF1), caused by mutation of the tumour-suppressor gene encoding neurofibromin, is an autosomal dominant disorder affecting various organs.

Aim. The aim of the study was to discuss the clinical symptoms of NF1 based on seven cases of the disease with regard to the literature.

Material and methods. We analyzed retrospectively patients with NF1 (4 females and 3 males) aged 19-52 who were treated at Endocrinology Department between 2003 and 2017. The diagnosis was made in childhood (4 patients) or in adolescence (3 cases), based on clinical symptoms and genetic tests.

Results. Five patients had a positive family history of NF1, two cases represented spontaneous mutation. All of analyzed subjects presented café-au-lait spots and neurofibromas on the body. We observed neurological disorders such as: epilepsy (2 patients), Arnold-Chiari malformation (1 man), benign brain neoplasms (2 persons). Optic gliomas appeared in two cases. Tumours were also found in other organs, including the uterus (2 women), the lung, the adrenals, the pituitary and the parathyroid gland (with signs of primary hyperparathyroidism) – each tumour in another patient. Four subjects suffered from cognitive impairment. Skeletal manifestations of neurofibromatosis type 1 such as scoliosis (1 man) and short stature (6 patients) have been noted as well. Five patients presented thyroid disorders – hypothyroidism due to Hashimoto’s disease (4 patients), toxic nodular goiter (1 woman). Other clinical symptoms e.g. vitiligo, alopecia areata and coarctation of aorta have also been found.

Conclusions. Variety of clinical symptoms causes that NF1 still remains a diagnostic and management challenge for many physicians. Therefore, multidisciplinary approach is needed to optimize patients’ treatment.

Open access
Modern methods of treatment of post-traumatic bone defects in Department of Trauma Surgery and Emergency Medicine, Medical University of Lublin

References 1. Marczyński W. Współczesne poglądy patogenetyczne a biologia zaburzeń zrostu kości długich. Ortop Traumatol. 2006;3:7-14. 2. Axelrad T, Kakar S, Einhorn T. New technologies for the enhancement of skeletal repair. Injury Int J Care Injured. 2007;38(Suppl.1):49-62. 3. Hamera-Słynarska M. Farmakologiczne wspomaganie gojenia złamań. Ortop Traumatol Rehab. 2000;3:11-4. 4. Belcarz A, Ginalska G, Pycka T, et al. Application of β-1,3-glucan in production of ceramics-based elastic composite

Open access
Long-term effects of reproductive period on mineralized tissues (bones and teeth) in peri- and postmenopausal women

. 2001;153:1166-72. 26. Bałczewska E. Analiza stanu uzębienia u kobiet z chorobami przyzębia i obniżoną gęstością mineralną kości. Czas Stomat. 2005,58(12):863-70. 27. Kunchur R, Goss AN. The oral health status of patients on oral bishosphonates for osteoporosis. Aust Dent J. 2008;53(4):354-7. 28. Drozdowska B, Pluskiewicz W, Michno M. Tooth count inederly women in relation to their skeletal status. Maturitas. 2006;55:126-31. 29. Kribbs PJ, Smith DE, Chesnut CH III. Oral findings in osteoporosis. Part 2

Open access
Effect of creatine ethyl ester supplementation and resistance training on hormonal changes, body composition and muscle strength in underweight non-athlete men

., Louis M., Theisen D., Nielens H., Dehoux M., Thissen J.P., Francaux M. (2005) Increased IGF mRNA in human skeletal muscle after creatine supplementation. Med. Sci. Sports Exerc. , 37(5): 731-736. DOI: 10.1249/01.MSS.0000162690.39830.27. 15. Farshidfar F., Pinder M.A., Myrie S.B. (2017) Creatine supplementation and skeletal muscle metabolism for building muscle mass-review of the potential mechanisms of action. Curr. Protein. Pept. Sci., 18(12): 1273-1287. DOI: 10.2174/1389203718666170606105108. 16. Gualano B., Macedo A.R., Alves C.R.R., Roschel H

Open access
The effect of 30-m repeated sprint exercise on muscle damage indicators, serum insulin-like growth factor-Iand cortisol

., 62: 50-58. 4. Berg U., Gustafsson T. Sundberg C.J., Kaijser L., Carlsson-Skwirut C., Bang P. (2007) Interstitial IGF-I in exercising skeletal muscle in women. Eur. J. Endocrinol ., 157: 427-435. 5. Brancaccio P., Maffulli N., Limongelli F.M. (2007) Creatine kinase monitoring in sport medicine. Br. Med. Bull. , 81-82: 209-230. 6. Buono M.J., Yeager J.E., Hodgon J.A. (1986) Plasma adrenocorticotropin and cortisol responses to brief high-intensity exercise in humans. J. Appl. Physiol ., 61: 1337-1339 7. Cappon J., Brasel J.A., Mohan S

Open access
Effects of military basic training on VO2max, body composition, muscle strength and neural responses in conscripts of different aerobic condition

.F., Gordon S.E., Harman E.A., Deschenes M.R., Reynolds K., Newton R.U., Triplett N.T., Dziados J.E. (1995) Compatibility of high-intensity strength and endurance training on hormonal and skeletal muscle adaptations. J. Appl. Physiol., (Bethesda, Md : 1985), 78: 976-989. 9. Kyröläinen H. Komi P.V. (1994) Neuromuscular performance of lower limbs during voluntary and reflex activity in power – and endurance-trained athletes. Eur. J. Appl. Physiol. and Occup. Physiol., 69: 233-239. 10. Kyröläinen H., Santtila M., Nindl B.C., Vasankari T. (2010) Physical fitness

Open access
The Use of Neuromuscular Electrical Stimulation of the Lower Limbs Skeletal Muscles in Cardiac Rehabilitation of Patients With Chronic Heart Failure

;79:357-362 6. Dal Corso S,Napolis L, Malaguti C. et al. Skeletal muscle structure and function in response to electrical stimulation in moderately impaired COPD patients. Respir. Med, 2007; 101:1236-1243 7. Deley G, Eicher JC, Verges B. et al. Do low-frequency electrical myostimulation and aerobic training similarly improve performance in chronic heart failure patients with different exercise capacities? J Rehabil Med, 2008; 40:219-224 8. Dobsak P,Navakova M, Fiser B. et al. Electrical stimulation of skeletal muscles. An alternative to aerobic

Open access
Changes of Serum Concentrations of Alkaline Phosphatase and Metalloproteinase-9 in an Ovariectomized Wistar Rat Model of Osteoporosis

References 1. Szulc P, Mary LB. Overview of osteoporosis: Epidemiology and clinical management. Vertebral Fracture Initiative Resource Document PART I. International Osteoporosis Foundation; 2011. 2. Senthilkumar K, Venkatesan J, Kim S. Marine derived natural products for osteoporosis. Biomed Prev Nutr. 2014;4(1):1-7. 3. Turner R, Riggs B, Spelsberg T. Skeletal effects of estrogen. Endocr Rev. 1994;15(3):275-300. 4. Walsh JS, Eastell R. Role of estrogen in the age-related decline in bone microstructure. Clin Endocrinol Metab J. 2013

Open access