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Increased breathing resistance compromises the time course of rhythmical forearm movements—a pilot study

REFERENCES 1. Gosselink R, Troosters T, Decramer M. Peripheral muscle weakness contributes to exercise limitation in COPD. Am J Respir Crit Care Med 1996;153:976-80. 2. Clark CJ, Cochrane LM, Mackay E, Paton B. Skeletal muscle strength and endurance in patients with mild COPD and the effects of weight training. Eur Respir J 2000;15:92-7. 3. Debigaré R, Maltais F. The major limitation to exercise performance in COPD is lower limb muscle dysfunction. J Appl Physiol 2008; 105: 751-3. 4. Donaldson AV, Maddocks M, Martolini D, Polkey MI, Man WD

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Lipodystrophy HIV-related and FGF21: A new marker to follow the progression of lipodystrophy?

, fasting triglycerides, body mass index, and waist-to-hip ratio [ 27 ] . In a recent paper, it has been demonstrated that FGF-21 mRNA expression is increased in skeletal muscle in patients with HIV-related lipodystrophy and that FGF-21 mRNA expression in muscle is associated with decreased limb fat mass, increased waist-to-hip ratio, and increased triglycerides [ 28 ] . Another well-conducted study found highly significant correlation between FGF21 levels and indicators of altered fat distribution in lipodystrophy and a positive relationship between FGF21 levels and

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Left Ventricular Cleft Detected by Transthoracic Echocardiography in a Patient with Autosomal Dominant Polycystic Kidney Disease

-370. 6. Hadimeri H, Caidahl K, Bech-Hanssen O, Nyberg G. Echocardiographic findings in kidney transplant patients with autosomal dominant polycystic kidney disease. Scand J Urol Nephrol 2009; 43(5): 416-419. 7. Catapano F, Pancaldi S, Pace Napoleone C, et al . An unusual cardiac manifestation in autosomal dominant polycystic kidney disease. Case Rep Nephrol 2012; 2012: 978170. 8. Boulter C, Mulroy S, Webb S, et al . Cardiovascular, skeletal, and renal defects in mice with a targeted disruption of the Pkd1 gene. Proceed Nat Acad SciUSA 2001; 98

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Nutrition in chronic obstructive pulmonary disease: A review

Nutr 2012; 95: 1385–95. 11. Hallin R, Koivisto-Hursti UK, Lindberg E, Janson C. Nutritional status, dietary energy intake and the risk of exacerbations in patients with chronic obstructive pulmonary disease (COPD). Respir Med 2006; 100: 561–7. 12. Langen RC, Gosker HR, Remels AH, Schols AM. Triggers and mechanisms of skeletal muscle wasting in chronic obstructive pulmonary disease. Int J Biochem Cell Biol 2013; 45: 2245–56. 13. Remels AH, Gosker HR, Langen RC, Schols AM. The mechanisms of cachexia underlying muscle dysfunction in COPD. J Appl

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PreSSUB II: The prehospital stroke study at the Universitair Ziekenhuis Brussel II

financing of musculo skeletal and neuro rehabilitation in Belgium]. 2007. 27. De Wit L, Putman K, Devos H, Brinkmann N, Dejaeger E, De Weerdt W, et al. Five-year mortality and related prognostic factors after inpatient stroke rehabilitation: A European multi-centre study. J Rehabil Med 2012;44:547-52. 28. Putman K, De Wit L, Schupp W, Baert I, Brinkmann N, Dejaeger E, et al. Variations in follow-up services after inpatient stroke rehabilitation: A multicentre study. J Rehabil Med 2009;41:646-53. 29. De Wit L, Putman K, Schuback B, Komarek A, Angst F, Baert I, et al

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Differentiating hyperlactatemia type A from type B: How does the lactate/pyruvate ratio help?

activity in rat skeletal muscle: effects on plasma lactate. Shock 1996; 6: 89-94. 8856841 10.1097/00024382-199608000-00002 Vary TC Sepsis-induced alterations in pyruvate dehydrogenase complex activity in rat skeletal muscle: effects on plasma lactate Shock 1996 6 89 – 94 12 Levy B, Gibot S, Franck P, Cravoisy A, Bollaert PE. Relation between muscle Na+K+ ATPase activity and raised lactate concentrations in septic shock : a prospective study. Lancet 2005; 365: 871-5. 10.1016/S0140-6736(05)71045-X 15752531 Levy B Gibot S Franck P Cravoisy A

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Ascorbic acid improves renal microcirculatory oxygenation in a rat model of renal I/R injury

-1271. 37. Karaman A, Turkmen E, Gursul C, Tas E, Fadillioglu E. Prevention of renal ischemia/reperfusion-induced injury in rats by leflunomide. Int J Urol 2006; 13: 1434-41. 38. Tyml K, Li F, Wilson JX. Delayed ascorbate bolus protects against maldistribution of microvascular blood flow in septic rat skeletal muscle. Crit Care Med 2005; 33: 1823-8. 39. Wang P, Zhu Q, Wu N, Siow YL, Aukema HOK. Tyrosol attenuates ischemia-reperfusion-induced kidney injury via inhibition of inducible nitric oxide synthase. J Agric Food Chem 2013; 17; 61

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The role of heat shock proteins in kidney disease

HSP 72 stress response of monocytes from patients on hemodialysis in impaired Nephrol Dial Transplant 2009 24 2838 46 19 Gowe AV, McArdle A, Mc Ardle F, Pattwell DM, Bell GM, Kemp GJ, et al . Markers of oxidative stress in the skeletal muscle of patients on hemodialysis. Nephrol Dial Transplant 2007; 22: 1177-83. 10.1093/ndt/gfl721 Gowe AV McArdle A Mc Ardle F Pattwell DM Bell GM Kemp GJ et al. Markers of oxidative stress in the skeletal muscle of patients on hemodialysis Nephrol Dial Transplant 2007 22 1177 83 20 Arbriter K, Bidmon B, Endenmann M

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Heterogeneity of aortic smooth muscle cells: A determinant for regional characteristics of thoracic aortic aneurysms?

Lyle JS Hoechstetter L Rose PS Levy HP et al Prevalence of aortic root dilation in the Ehlers-Danlos syndrome Genet Med 2002 4 112 – 7 7 Loeys BL, Chen J, Neptune ER, Judge DP, Podowski M, Holm T, et al . A syndrome of altered cardiovascular, craniofacial, neurocognitive and skeletal development caused by mutations in tgfbr1 or tgfbr2. Nat Genet 2005; 37: 275-81. 10.1038/ng1511 Loeys BL Chen J Neptune ER Judge DP Podowski M Holm T et al A syndrome of altered cardiovascular, craniofacial, neurocognitive and skeletal development

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Therapeutic approaches of obstructive sleep apnea in China

failure, arrythmia, stroke, and impaired glucose tolerance.[ 2 ] Therefore, accurate diagnosis and treatment of OSA is strongly recommended so as to improve quality of life and reduce the associated morbidity and mortality. Many factors can give rise to OSA, such as skeletal malformations, soft tissue crowding, and respiratory instability. Sometimes these factors are interacting. Thus, from a clinical point of view, the patient-directed therapeutic approaches should be individually scheduled based on personalized situation. The current treatment of OSA in China

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