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of an Ascaris inhibitor for pepsin and cathepsin E. Eur. J. Biochem., 253: 804 - 809. DOI: 10.1046/j.1432-1327.1998.2530804.x KLIMENKO, V., ĶĔNIŅA, V. (1971): On the influence of phylogeny and ecology on some biochemical mechanisms of helminth adaptation. Latvijas Zinatnu akademijas vestis, 11: 93 - 96. (In Russian) KNOX, D.P. (2007): Proteinase inhibitors and helminth parasite infection. Parasite Immunol., 29: 57 - 71. DOI: 10.1111/j.1365-3024.2006.00913.x KUZ’MINA, V.V., IZVEKOVA, G.I., KUPERMAN, B.I. (2000): Peculiarities of nutrition physiology in cestodes and

.L., MONEO, I. (2004): Several allergens from Anisakis simplex are highly resistant to heat and pepsin treatments. Parasitol. Res., 93: 248 - 251. DOI: 10.1007/s00436-004-1099-3 CARBALLEDA-SANGIAO, N., OLIVARES, F., RODRIGUEZ-MAHILLO, A.I., CARECHE, M., TEJADA, M., MONEO, I., GONZALEZ-MUNOZ, M. (2014): Identifi cation of autoclave-resistant Anisakis simplex allergens. J. Food Prot., 77: 605 - 609. DOI: 10.4315/0362-028X.JFP-13-278 CHAI, J.Y., DARWIN MURRELL, K., LYMBERY, A.J. (2005): Fish-borne parasitic zoonoses: status and issues. Int. J. Parasitol., 35: 1233 -1254. DOI

stock Swiss mice by 1 % pepsin/1 % HCl digestion ( Dunn & Wright, 1985 ). Two groups of seven animals received GA at a dose of 0.5 and 1 mg/ kg by intraperitoneal injection once every other day to give a total of 3 doses. Treatment started at 7 th day p.i. The remaining mice served as a control infected group. Additional seven infected mice received only DMSO. At 35 days p.i., the animals were euthanized, and muscle larvae were recovered and counted as described before ( Dunn & Wright, 1985 ). Similar parts of the diaphragm and thigh muscles were obtained and