Akinjide M. Akinnuga, Olubayode Bamidele and Anthony J. Adewumi
. Nevin KG, Rajamohan T. Virgin coconut oil supplemented diet increases the antioxidant status in rats. Food Chemistry 2006; 99: 260-6.
10. Kiran G, Nandini CD, Ramesh HP, et al. Progression of early phase diabetic nephropathy in streptozotocin-induced diabetic rats. Evaluation of various kidney-related parameters. Indian J Exp Biol 2012; 50: 133-40.
11. Grover JK, Yadav SP, Vats V, et al. Effect of feeding Murrayakoeingii and Brassica juncea diet on kidney functions and glucose levels in streptozotocin diabetic mice. J Ethnopharmacol 2003; 85(1): 1
Victoria Ancuța Rus, Florina Ruța, Maria Sălcudean, Monica Tarcea, Costela Șerban, Călin Avram, Iustinian Simion and Theodora Benedek
European Region. Available at: http://www.euro.who.int/__data/assets/pdf_file/0008/185903/e96816.pdf .
4. World Health Organization. Global Health Observatory (GHO) data: Raised blood pressure. Available at: http://www.who.int/gho/ncd/risk_factors/blood_pressure_prevalence_text/en .
5. Blumenthal JA, Babyak, MA, Hinderliter, A et al. Effects of the DASH diet alone and in combination with exercise and weight loss on blood pressure and cardiovascular biomarkers in men and women with high blood pressure: the ENCORE study. Arch Intern Med . 2010
Stela Mariana Al Hussein, Hussam Al Hussein, Camil Eugen Vari, Nicoleta Todoran, Hamida Al Hussein, Adriana Ciurba and Maria Titica Dogaru
6. Rosenfield R. Ovarian and adrenal function in polycystic ovary syndrome. Endocrinol Metab Clin North Am. 1999;28:265-293.
7. Rosenfield R. Polycystic ovary syndrome and insulin resistant hyperinsulinemia. J Am Acad Dermatol. 2001;45:S95-S104.
8. Davidovici BB, Wolf R. The role of diet in acne: facts and controversies. Clin Dermatol. 2010;28:12–16.
9. Hoyt G, Hickey MS, Cordain L. Dissociation of the glycaemic and insulinaemic responses to whole and skimmed milk. Br J Nutr. 2005;93(2):175-177.
10. Hoppe C, Molgaard C, Michaelsen KF. Cow
Ianosi Edith Simona, Comes Alexandra and Jimborean Gabriela
tolerance. N Engl J Med. 2001;344:1343-1350.
41. Wadden TA, Sternberg JA, Letizia KA, Stunkard AJ. Treatment of obesity by very low calorie diet, behaviour therapy and combination: a 5 year experience. Int J Obes. 1989;13S:39-46.
42. Dixon JB, et al. Adjustable gastric banding and conventional therapy for type 2 diabetes: a randomized controlled trial. JAMA. 2008;299:316–323.
43. Freitas A, Sweeney JF. Bariatric Surgery. In: B. Banerjee. Nutritional Management of Digestive Disorders. Boca Raton, FL: CRC Press. 2010;327–342.
44. Shikora SA, Kim J
Objective: Our study aim to evaluate the characteristics of nutrition behavior at teenagers from Moldova’s rural area schools in order to make a profile of risk behavior related to diet and to implement efficient community intervention programs.
Methods: We studied in 2014 the daily frequency consumption of the main food groups, in a sample of 1236 students of 7th-12th class, from rural localities of the Republic of Moldova, using the questionnaire method.
Results: The teenagers had consumed on the day preceding the interview, cereals and bakery products – 99.2%, fruits – 86.7%, cooked vegetables – 72.6%, meat dishes – 72.1%, potatoes – 50.3%, dairy products – 46.9%, raw vegetables – 44.7%, eggs – 25.3%, fish – 19.4%, raw fruits juice – 14.8%, legumes – 12.5% of students. From those investigated 52.6% have consumed food with high fat content higher than recommended levels. Regarding the food groups present in the menu we registered a satisfactory level of consumption by the students for cereals and bakery products, fruits, cooked vegetables and meat dishes; relatively satisfactory - the consumption of dairy products, raw vegetables and potatoes; unsatisfactory level for the consumption of eggs, fish and legumes.
Conclusions: We sustain the idea that it is important to constantly evaluate the risk factors related to the inadequate diet at teenagers and to implement efficient community intervention in order to prevent nutrition related diseases that can occur.
Sutharinee Likitnukul, Sarinee Kalandakanond-Thongsong and Sumpun Thammacharoen
Center, Mahidol University, and individually housed in conventional hanging cages with stainless steel wire mesh floors (33 cm × 18 cm × 20 cm) under standard condition(12 h/12 h light/dark cycle, 22 ± 1°C). All rats were allowed to acclimatize to the experimental condition for at least 2 weeks before starting the experiment. The rats were fed standard diet rat chow (#082; Perfect Companion Group Ltd., Samutprakarn, Thailand; protein 24%, carbohydrate 42%, fat 4.5%, energy 3.04 kcal/g, energy from fat 13%) and water ad libitum . During the 6 weeks of obesity
Nemanja Jovicic, Ilija Jeftic, Marina Miletic Kovacevic, Irena Tanaskovic, Nebojsa Arsenijevic, Miodrag L. Lukic and Nada Pejnovic
2 enhances high - fat diet -induced visceral adiposity and inflammation in BALB/c mice [Delecija gena za ST2 promoviše gojaznost i inflamaciju u visceralnom adipoznom tkivu BALB/c miševa na dijeti sa visokim sadržajem masti]. Serb J Exp Clin Res 2013; 14(4): 155 -160.
25. Donnelly KL, Smith CI, Schwarzenberg SJ, Jessurun J, Boldt MD, Parks EJ. Sources of fatty acids stored in liver and secreted via lipoproteins in patients with nonalcoholic fatty liver disease. J Clin Invest. 2005;115(5):1343-51.
26. Langin D. Adipose tissue lipolysis as a metabolic
Ilija Jeftic, Marina Miletic-Kovacevic, Nemanja Jovicic, Jelena Pantic, Nebojsa Arsenijevic, Miodrag L. Lukic and Nada Pejnovic
accelerates high-fat diet induced obesity and amplifies inflammation in adipose tissue and pancreatic islets. Diabetes 2013,62:1932-44.
19. Weigert J, Neumeier M, Wanninger J, Bauer S, Farkas S, Scherer MN et al. Serum galectin-3 is elevated in obesity and negatively correlates with glycosylated hemoglobin in type 2 diabetes. J Clin Endocrinol Metab 2010:95:1404-11
20. Rhodes DH, Pini M, Castellanos KJ, Montero-Melendez T, Cooper D, Perretti M et al. Adipose tissue-speific modulation of galectin expression in lean and obese mice: evidence for regulatory function
Saracut Claudiu, Molnar Calin, L Farczádi, L Vlase, Tero-Vescan Amelia, Todoran Nicoleta and Copotoiu Constantin
Objectives: The aim of the study was to determine the level of secondary bile acids (SBA) in the diets and feces of mice and the variation of amount ingested/excreted if these SBA are administered as monotherapy or in 1:1 dose.
Methods: The mice were divided into 4 groups and fed for 140 days with different diets. The control lot received a normal diet and the others received diets supplemented with 0.25% deoxycholic acid (DCA), 0.25% lithocholic acid (LCA) and 0.125% DCA+0.125% LCA. After 140 days, the mice feces were collected and homogenized to obtain a mixture for each lot from which the determinations of the studied SBA were performed. For the mice food evaluation, portions of 10 g from each of the 4 diets were subjected to the SBA determination.
Results: The daily ingestion over more than 4 months of DCA or LCA added to the diet and administered as monotherapy determine a significantly increase of the SBA eliminated into the feces (the DCA level was 11x higher, and of the LCA 233x higher). If half of the LCA dose is replaced with DCA, the level of LCA in the feces gets comparable with that of the DCA (their combined amounts represents only 13x higher increase of these two bile acids in feces).
Conclusions: The simultaneous ingestion and excretion of DCA and LCA can be considered as a particular situation ruled by endogenous mechanisms. This behavior represents an important observation, knowing that the bile acids effects in the colorectal cancer are dose dependent.