Ana B. Segarra, Isabel Prieto, Magdalena Martinez-Canamero, Jose-Ignacio Ruiz-Sanz, M. Begona Ruiz-Larrea, Marc De Gasparo, Inmaculada Banegas, Stefan Zorad and Manuel Ramirez-Sanchez
-dye binding. Anal Biochem 72, 248–254, 1976.
Chang GQ, Karatayev O, Ahsan R, Gaysinskaya V, Marwil Z, Leibowitz SF. Dietary fat stimulates endogenous enkephalin and dynorphin in the paraventricular nucleus: role of circulating triglycerides. Am J Physiol Endocrinol Metab 292, E561–E570, 2007.
Chang GQ, Karatayev O, Barson JR, Chang SY, Leibowitz SF. Increased encephalin in brain of rats prone to overconsuming a fat-rich diet. Physiol Behav 101, 360–369, 2010.
Corder G, Castro DC, Bruchas MR, Scherrer G. Endogenous and exogenous opioids in pain. Annu Rev
Paulina Pidikova, Pavel Svitok and Iveta Herichova
human liver disease and has antifibrotic activity in the bile-duct-ligated rat. Clin Sci (Lond) 117, 375–386, 2009.
Lyngso C, Erikstrup N, Hansen JL. Functional interactions between 7TM receptors in the renin-angiotensin system--dimerization or crosstalk? Mol Cell Endocrinol 302, 203–212, 2009.
Mao C, Liu R, Bo L, Chen N, Li S, Xia S, Chen J, Li D, Zhang L, Xu Z. High-salt diets during pregnancy affected fetal and offspring renal renin-angiotensin system. J Endocrinol 218, 61–73, 2013.
Matsusaka T, Niimura F, Shimizu A, Pastan I, Saito A, Kobori H
Heba A. Abdel-Hamid, Mona M. I. Abdalla, Nagwa M. Zenhom and Rasha F. Ahmed
of the Y2-receptor promotes secretion of GLP-1 and improves glucose tolerance. Mol Metab 2, 142–152, 2013.
Chao PT, Yang L, Aja S, Moran TH, Bi S. Knockdown of NPY expression in the dorsomedial hypothalamus promotes development of brown adipocytes and prevents diet-induced obesity. Cell Metab 13, 573–583, 2011.
Chatree S, Churintaraphan M, Nway N, Sitticharoon C. Expression of Y2R mRNA expression in subcutaneous and visceral adipose tissues compared between normal weight and obese female subjects. Siriraj Medical Journal 65, 163–167, 2017
Joanna Rog, Kaja Karakuła, Agnieszka Próchnicka and Hanna Karakula-Juchnowicz
Introduction: Schizophrenia is a chronic mental disease which significantly affects functioning and quality of life of patients. Lifestyle, including irregular eating habits, is a factor possibly intensifying symptoms of the disease and unwanted effects of pharmacotherapy. Due to positive effect on metabolic parameters demonstrated in numerous studies and participation in structure and functioning of central nervous system, omega-3 essential unsaturated fatty acids (EFAs) are the suggested form of schizophrenia cotheraphy.
Aim: The purpose of this paper was to evaluate EFAs (especially omega-3 family) consumption by individuals with schizophrenia and comparing contents of these acids in the diets of female and male patients.
Method: A study was conducted with the participation of 32 patients, recruited in the Psychiatric Outpatient Department of the Independent Public Clinical Hospital No. 1 in Lublin, with diagnosed schizophrenia. Data concerning the intake of EFAs was acquired in the course of 24-hour diet recall.
Results: Average intake of omega-3 EFAs in the examined population was 2.40 ± 2.85 g, of which only 201.6 ± 501.5 mg was constituted by EPA and DHA acids. The intake of omega-3 EFAs did not differ between men and women groups (p>0.05). Food rations of the 91% responders were characterised with the intake of EPA and DHA acids below the adequate intake level (AI).
Conclusions: Patients suffering from schizophrenia consume insufficient amounts of EPA and DHA acids. Evidence suggests that optimum intake of omega-3 EFAs could bring significant benefits for this group of patients. People suffering from schizophrenia should be attended with the care of a dietician, who will choose optimum strategy for supplying sufficient amount of nutrients in the diet.
Joanna Tomaka, Hanna Karakuła-Juchnowicz, Justyna Morylowska-Topolska, Michał Dzikowski, Dariusz Juchnowicz, Marta Flis, Aleksandra Siek and Michał Próchnicki
al. Diagnosis of Non-Celiac Gluten Sensitivity (NCGS): The Salerno Experts' Criteria. Nutrients. 2015;7(6):4966-77.
28. Cooper BT., Holmes GK., Ferguson R., Thompson RA., Allan RN., Cooke WT. Gluten-sensitive diarrhea without evidence of celiac disease. Gastroenterology. 1980; 79: 801-806
29. Digiacomo, D.V., Tennyson, C.A., Green, P.H., & Demmer, R.T. Prevalence of gluten-free diet adherence among individuals without celiac disease in the USA: Results from the continuous national health and nutrition examination survey 2009
Carlos Eduardo C. Martins, Vanessa B. de Sousa Lima, Henrique Quintas T. Ribeiro and Julio Tirapegui
polyherbal formulation in steptozotocin induced hyperglycemic rats. Anc Sci Life 34, 23–32, 2014.
Ghanbari-Niaki A, Saghebjoo M, Hedayati M. A single session of circuit-resistance exercise effects on human peripheral blood lymphocyte ABCA1 expression and plasma HDL-C level. Regul Pept 166, 42–47, 2011.
Guido S, Joseph T. Effect of chemically different calcium antagonists on lipid profile in rats fed on a high fat diet. Indian J Exp Biol 30, 292–294, 1992.
Hinault C, Mothe-Satney I, Gautier N, Van Obberghen E. Amino acids require glucose to enhance
., Ibarra Jato M., Barroso García A., González Tejón S., Tajada Vitales C., Díaz Mújica B., Viñas Cabrera L., Sanchís Catalán R., Salvador Barbarroja T. The effectiveness of a program of physical activity and diet to modify cardiovascular risk factors in patients with severe mental illness after 3-month follow-up: CAPiCOR randomized clinical trial. Eur Psychiatry. 2015; 30(8): 1028-36.
7. Kraeuter A.K., Loxton H., Lima B.C., Rudd D., Sarnyai Z. Ketogenic diet reverses behavioral abnormalities in an acute NMDA receptor hypofunction model of schizophrenia. Schizophr
Ayodele Olufemi Morakinyo, Titilola Aderonke Samuel and Daniel Abiodun Adekunbi
insulin resistance in a sample of young, nondiabetic Black Americans. Am J Hypertens 12, 747-756, 1999.
Irondi EA, Oboh G, Akindahunsi AA. Antidiabetic eff ects of Mangifera indica Kernel Flour-supplemented diet in streptozotocin-induced type 2 diabetic in rats. Food Sci Nutr 4, 828-839, 2016.
Joshi RL, Lamothe B, Cordonnier N, Mesbah K, Monthioux E, Jami J, Bucchini D. Targeted disruption of the insulin receptor gene in the mouse results in neonatal lethality. Embo J 15, 1542-1547, 1996.
Kisters K, Gremmler B, Kozianka J
YP. Inhibition of lipoprotein-associated phospholipase A2 ameliorates inflammation and decreases atherosclerotic plaque formation in ApoE-deficient mice. PLoS ONE 6, e23425, 2011.
Wihastuti TA, Sargowo D, Tjokroprawiro A, Permatasari N, Widodo MA, Soeharto S. Vasa vasorum anti-angiogenesis through H2O2, HIF-1α, NF-κB, and iNOS inhibition by mangosteen pericarp ethanolic extract (garcinia mangostana linn) in hypercholesterol-diet-given rattus norvegicus wistar strain. Vasc Health Risk Manag 10, 523–531, 2014.
Wihastuti TA, Heriansyah H, Soraya M
Małgorzata Futyma-Jędrzejewska, Hanna Karakuła-Juchnowicz and Ewelina Drzał
Pregnancy, childbirth and motherhood are new situations for women and require adjustment. Women suffering from schizophrenia require special attention due to the course of the disease. Physiological changes that occur in the body during pregnancy may be unacceptable for women suffering from schizophrenia. They may delusively deny the existence of the pregnancy, lead an unhealthy lifestyle (stimulants, poor diet, lack of gynaecological check-ups), which in turn causes an increased risk of complications. In the research conducted so far, it has been proven that three kinds of complications are associated with schizophrenia: complications concerning pregnancy itself (bleeding, diabetes, Rh-incompatibility, pre-eclampsia), intrauterine growth restriction (low birth weight, congenital malformations, small head circumference) and complications regarding labour (uterine atony, asphyxia, emergency Caesarean section). The course of the labour itself in this specific group of patients has not yet been sufficiently examined. It has also been proven that perinatal complications are one of the factors determining an increased risk of schizophrenia.