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The relationship between labor pain management, cortisol level and risk of postpartum depression development: a prospective nonrandomized observational monocentric trial

Behav Immun 2015; 49: 86-93. doi: 10.1016/j.bbi.2015.04.012 6. Corwin EJ, Brownstead J, Barton N, Heckard S, Morin K. The impact of fatigue on the development of postpartum depression. J Obstet Gynecol Neonatal Nurs 2005; 34: 577-586. doi: 10.1177/0884217505279997 7. Beck CT. Postpartum depression: it isn’t just the blues. Am J Nurs 2006; 106: 40-50 8. Hay DF, Pawlby S, Waters CS, Sharp D. Antepartum and postpartum exposure to maternal depression: different effects on different adolescent outcomes. J Child Psychol

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Predictors of Left Ventricular Remodeling Post Acute Myocardial Infarction. Protocol for a Clinical Study


Heart failure is a clinical syndrome that appears as a consequence of a structural disease, and the most common cause of left ventricular systolic dysfunction results from myocardial ischemia. Cardiac remodeling and neuroendocrine activation are the major compensatory mechanisms in heart failure. The main objective of the study is to identify the association between serum biomarkers illustrating the extent of myocardial necrosis (highly sensitive troponin as-says), left ventricular dysfunction (NT-proBNP), and systemic inflammatory response (illustrated via serum levels of hsCRP and interleukins) during the acute phase of a myocardial infarction, and the left ventricular remodeling process at 6 months following the acute event, quantified via speckle tracking echocardiography. The study will include 400 patients diagnosed with acute myocardial infarction without signs and symptoms of heart failure at the time of enrollment that will undergo a complex clinical examination and speckle tracking echocardiography. Serum samples from the peripheral blood will be collected in order to determine the inflammatory serum biomarkers. After 6 months, patients will be divided into 2 groups according to the development of ventricular remodeling, quantified by speckle tracking echocardiography: group 1 will consist of patients with a remodeling index lower than 15%, and group 2 will consist of patients with a remodeling index higher than 15%. All clinical and imaging data obtained at the baseline will be compared between these two groups in order to determine the features associated with a higher risk of deleterious ventricular remodeling and heart failure.

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Immune Thrombocytopenia in an Adolescent with Hashimoto’s Thyroiditis – Case Report

REFERENCES 1. Sugiura T, Yamamoto K, Murakami K. Immune Thrombocytopenic Purpura Detected with Oral Hemorrhage: a Case Report. J Dent Shiraz Univ Med Sci . 2018;19:159-163. 2. Hazzan R, Mukamel M, Yacobovich J. Risk factors for future development of systemic lupus erythematosus in children with idiopathic thrombocytopenic purpura. Pediatr Blood Cancer . 2006;47:657-659. 3. Kuhne T. Diagnosis and management of immune thrombocytopenia in childhood. Hamostaseologie . 2017;37:36-44. 4. Seidel MG. Autoimmune and other cytopenias in primary

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Toxic Megacolon – the Complication of an Infection with Clostridium difficile after an Odontogenic Abscess. Case Presentation

MA, Bhatnagar SR, Feldman L, et al. Development and validation of a clinical risk calculator for mortality after colectomy for fulminant Clostridium difficile colitis. Journal of Trauma and Acute Care Surgery. 2019. [Epub ahead of print]

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Noninvasive Imaging Biomarkers of Vulnerable Coronary Plaques – a Clinical Update

for atherosclerosis imaging in drug development. Nat Rev Drug Discov . 2008;7:517-529. 41. Amirbekian V, Lipinski MJ, Briley-Saebo KC, et al. Detecting and assessing macrophages in vivo to evaluate atherosclerosis noninvasively using molecular MRI. Proc Natl Acad Sci . 2007;104:961-966. 42. McAteer MA, Schneider JE, Ali ZA, et al. Magnetic Resonance Imaging of Endothelial Adhesion Molecules in Mouse Atherosclerosis Using Dual-Targeted Microparticles of Iron Oxide. Arterioscler Thromb Vasc Biol . 2008;28:77-83. 43. Mitsumori LM, Hatsukami TS

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Sinonasal inverted papilloma – what’s new

. Am J Surg. 1985;150(4):406–13. 31. Lund VJ. Optimum management of inverted papilloma. J Laryngol Otol. 2000;114(3):194-7. 32. Dammann F, Pereira P, Laniado M, Plinkert P, Lowenheim H, Claussen CD. Inverted papilloma of the nasal cavity and the paranasal sinuses: using CT from primary diagnosis and follow-up. AJR Am J Roentgenol. 1999;172(2):543-8. 33. Stankiewicz JA, Girgis SJ. Endoscopic surgical treatment of nasal and paranasal sinus inverted papilloma. Otolaryngol Head Neck Surg. 1993;109(6):988–95. 34. Krouse JH. Development of a

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An Overview of Disease Burden, Mechanism, Traditional and Non-traditional Management of Type 2 Diabetes

: the CoLaus study. Diabet Med . 2012;29:190-197. 8. Bhargava A. A longitudinal analysis of the risk factors for diabetes and coronary heart disease in the Framingham Offspring Study. Popul Health Metr . 2003;1:3. 9. Helmrich SP, Ragland DR, Paffenbarger JR. Prevention of non-insulin-dependent diabetes mellitus with physical activity. Med Sci Sports Exerc . 1994;26:824-830. 10. Akram H. Tobacco use and development of diabetes: examination of the underlying mechanisms. J Ann Eu Med . 2018;6:1. 11. Al-Thani MH, Al-Thani AA, Al-Emadi AA, Al

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Fatal Hypocalcaemia Due to Hungry Bone Syndrome with Secondary Refractory HyperParathyroidism after Parathyroidectomy: A Case Report

]. The prevalence of HBS is contrarily reported in the literature, with figures ranging between 4-95% depending on several factors, i.e. primary or secondary parathyroid disease, geographic area [ 6 ]. The complication occurs more frequently after parathyroidectomy for renal hyperparathyroidism, where the clinical manifestation of HBS is more severe [ 7 ]. Such a case, with rapid fatal development shortly after parathyroidectomy, is presented here. The literature reports relatively few such situations, those reported generally concerning post-surgical treatment of SHPT

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