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apoptosis after glucocorticoid treatment. Am J Pathol. 1982; 109: 78-87. 4. Mitevska E, Kostovska N, Vasilevska K, Trajkov D. Stereological study of thymus after dexamethasone application. Maced Pharm Bull. 2004; 50(2): 25-28. 5. Mitevska E, Spiroski M. Qualitative histological analysis of adrenal cortex following application of medroxyprogesterone acetate. Maced J Med Sci. 2010; 3(2): 123-131. 6. Mitevska E, Spiroski M. Quantitative analysis of adrenal cortical histological alterations after application of medroxyprogesterone acetate. Maced J Med Sci. 2010; 3(4): 352

. Best Pract Res Clin Haematol. 2010; 23(1): 33-45. 10. Döhner H, Stilgenbauer S, Benner A, Leupolt E, Kröber A, Bullinger L, et al. Genomic aberrations and survival in chronic lymphocytic leukemia. N Engl J Med. 2000; 343(26): 1910-6. 11. Cimmino A, Calin GA, Fabbri M, Iorio MV, Ferra-cin M, Shimizu M, et al. miR-15 and miR-16 induce apoptosis by targeting BCL2. Proc Natl Acad Sci U S A. 2005; 102(39): 13944-9. 12. Klein U, Lia M, Crespo M, Siegel R, Shen Q, Mo T, et al. The DLEU2/miR-15a/16-1 cluster controls B cell proliferation and its deletion leads to chronic lym

, Bedognetti D, De Giorgi V et al. The immune-related role of BRAF in melanoma. Mol Oncol 2015; 9(1): 93–104. 14. Conrad WH, Swift RD, Biechele TL et al. Regulating the response to targeted MEK inhibition in melanoma: enhancing apoptosis in NRAS- and BRAF-mutant melanoma cells with Wnt/β-catenin activation. Cell Cycle. 2012; 11(20): 3724–30. 15. Rojas AM, Fuentes G, Rausell A et al. The Ras protein superfamily: evolutionary tree and role of conserved amino acids. J Cell Biol. 2012; 196(2): 189–201. 16. Baines AT, Xu D, Der CJ. Inhibition of Ras for cancer treatment: the

obstruction: immune cell infiltrate in the kidney. Kidney Int. 1988; 34: 487. 19. Kennedy WA, Buttyan R, Sawczuk IS, et al. Epidermal growth factor suppresses renal tubular apoptosis following ureteral obstruction. Urology. 1997; 49: 973–80. 20. Restrepo N, Kess-Folts D, Ding G. Tubulointerstitial macrophages, transforming growth factorbeta, and monocyte chemoattractant peptide (MCP)-1 after unilateral ureteral obstruction. J Am Soc Nephrol. 1993; 4: 826–33. 21. Carr MC. Anomalies and surgery of the ureteropelvic junction in children. In: Walsh PC, Retik BA, Vaughan ED

associated with positive D-dimer results in patients evaluated for pulmonary embolism. Academic emergency medicine: 2010; 17(6): 589–597. 13. Nguyen HB, Loomba M, Yang JJ, et al. Early lactate clearance is associated with biomarkers of inflammation, coagulation, apoptosis, organ dysfunction and mortality in severe sepsis and septic shock. Journal of Inflammation. 2010; 7: 6. 14. Barthwal M.S. Intrapleural fibrinolytic therapy in complicated parapneumonic effusion and empyema: present status. Indian journal of chest diseases and allied sciences 50.3 (2008): 277. 15. Satoshi

MN. Adiponectin Inhibits Nutrient Transporters and Promotes Apoptosis in Human Villous Cytotrophoblasts: Involvement in the Control of Fetal Growth. Biol Reprod. 2016; 94(5): 111. 32. Berner HS, Lyngstadaas SP, Spahr A, et al. Adiponectin and its receptors are expressed in bone forming cells. Bone 2004; 35: 842–9. 33. Lin YY, Chen CY, Chuang Ty, et al. Adiponectin receptor 1 regulates bone formation and osteoblast differentiation by GSK 3β/β-catein signaling in mice. Bone 2014; 64: 147–54. 34. Cekmez F, Aydemir G, Aydinoz S, et al. Low 25-hydroxyvitamin D level and

. Republic of Macedonia State Statistical Office. Book X. 2002; 10: 18-197. 10. Mograbi B, Bocciardi R, Bourget I, Juhel T, Farahi- Far D, Romeo G, et al. The sensitivity of activated Cys Ret mutants to glial cell line-derived neuritrophic factor is mandatory to rescue neuroectodermal cells from apoptosis. Mol Cell Biol. 2001; 21(20): 6719-6730. 11. Komminoth P. The role of ret proto-oncogene mutation analysis in the diagnosis of multiple endocrine neoplasia type 2 (MEN2) gene carriers and the discrimination of sporadic and familial medullary thyroid carcinomas and

development after estrogen loss: mediation by interleukin-6. Science. 1992; 257: 88–91. 21. Kameda T, Mano H, Yuasa T, Mori Y, Miyazawa K, Shiokawa M, et al. Estrogen inhibits bone resorption by directly inducing apoptosis of the bone-resorbing osteoclasts. The Journal of experimental medicine. 1997; 186: 489–95. 22. Horowitz MC. Cytokines and estrogen in bone: antiosteoporotic effects. Science.1993; 260: 626–7. 23. Passeri G, Girasole G, Jilka RL, Manolagas SC. Increased interleukin-6 production by murine bone marrow and bone cells after estrogen withdrawal. Endocrinology

. Goumenos DS, Tsamandas AC, Oldroyd S et al. Transforming growth factor-beta (1) and myofibroblasts: a potential pathway towards renal scarring in human glomerular disease. Nephron. 2001; 87(3): 240–8. 6. Goumenos DS, Kalliakmani P, Tsakas S, Papachristou E, Vlachojannis JG. Growth factors and apoptosis-related protein expression in human crescentic nephritis. Med Sci Monit. 2008; 14: 243–48. 7. Papasotiriou M, Kalliakmani P, Huang L et al. Does treatment with corticosteroids and cyclosporine reduce transglutaminase type 2 expression in the renal tissue of patients with

impairment and proliferation enhancement in cultured HPRT deficient neuroblastoma cells, a neuronal model of LND, has also been proposed [ 73 ]. Intrastriatal hypoxanthine administration to 60-day-old rats altered neuroenergetic parameters, resulting in ATP depletion and mitochondrial dysfunction and cell death by apoptosis, suggesting that these processes may be associated, at least in part, with neurological symptoms found in LND patients [ 74 ]. Deficit of other purine compounds due to HPRT defect is controversial, and altered nucleotide concentrations have been