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-abdominal infection is frequently extremely difficult to treat [ 1 ], and the mortality rate is reportedly high [ 2 ]. In an animal sepsis model of intra-abdominal infection, intra-abdominal fluid contained larger amounts of cytokines than seen in circulating blood [ 8 ]. These cytokines are continuously transferred into circulating blood, causing damage to the vascular endothelium of internal organs. In the lungs, this increases vascular permeability, increasing the volume of interstitial fluid and causing the appearance of diffuse infiltration on CT [ 9 ]. In particular, if

used, and measurements Data collected included demographic data (age, sex), comorbidities, source of sepsis, sodium levels on days one, three, and seven. The source of sepsis was confirmed via a routine sepsis workup that included radiological evidence, culture and sensitivity of blood, sputum, and urine, abdominal fluid drain culture, tissue culture, and cerebrospinal fluid (CSF) analysis and cultures for suspected cases. Various variables including demographic data, comorbidities, source of sepsis, and sodium levels on days 1, 3, and 7 were assessed against the