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Serving the Health Care and Leisure Needs of Ethnic Aged in Canada: Implications and Concerns

.pdf Paplauskas-Ramunas, A., & Université d’Ottawa Institut d’éducation physique. (1960). L’Éducation physique dans l’humanisme intégral (2e éd.). Ottawa, Ontario: Éditions de l’Université d’Ottawa. Physical Activity Resource Centre. (2013). Physical activity promotion for older adults: A step-by-step guide. Toronto, ON: Ophea. Retrieved from http://parc.ophea.net/resource/physical-activity-promotion-older-adults-step-step-guide Public Health Agency of Canada. (2010). Six Types of Cardiovascular Disease. Retrieved from http://www.phacaspc.gc.ca/cd-mc/cvd-mcv/cvd

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No Modification in Blood Lipoprotein Concentration but Changes in Body Composition After 4 Weeks of Low Carbohydrate Diet (LCD) Followed by 7 Days of Carbohydrate Loading in Basketball Players

oxidation, stimulate the immune system by suppression of lymphocyte proliferation, while reducing myocardial infarction, stroke, and death from CVD causes ( Lovejoy et al., 2002 ; Simopoulos, 2008 ). The most important PUFAs, eicosapentaenoic acid (EPA) and docosahexaenoic acid (DHA), which reduce the concentration of triglycerol in the blood, normalize blood pressure by increasing the level of prostacyclin, and they have an anticlotting effect ( Joris and Mensink, 2016 ). EPA and DHA acids have anti-inflammatory, anti-carcinogenic, and anti-sclerotic properties

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The contribution of paraoxonase 1 and myeloperoxidase to HDL-cholesterol functionality

Summary

The purpose of this study was to analyse the scientific evidence concerning the effects of two enzymes – paraoxonase 1 and myeloperoxidase – on the functions of HDL-cholesterol. It is well documented that disturbed circulating lipoproteins (a high total and high LDL-cholesterol, and low HDL-cholesterol) bring about atherosclerosis and an increased risk of cardiovascular disease (CVD) which is recognised as the main cause of death all around the world. In consequence, numerous studies have focused on procedures which will improve the plasma lipoproteins profile by decreasing the total cholesterol and the LDL-cholesterol (LDL-C) and increasing the HDL-cholesterol (HDL-C). However, the anti-atherogenic role of HDL-C has been challenged in studies showing that genetically elevated HDL-cholesterol does not offer protection against CVD. Moreover, it has been found that raising the circulating HDL-cholesterol fails to reduce atherosclerosis. The doubts concerning the protective role of HDL-C have been supported by in vitro studies which indicate that the HDL-C from patients with atherosclerosis does not have a protective action, but does stimulate inflammation and free radical synthesis. The above data suggests that HDL-C, commonly recognised as protective against atherosclerosis, in some circumstances becomes pro-atherogenic, and is thus dysfunctional. Our review focuses on two enzymes – paraoxonase 1 (PON1) and myeloperoxidase (MPO) – which markedly affect the properties of HDL-C and contribute to its anti – or pro-atherogenic activity. Moreover, the effects of the diet and physical activity on PON1 and MPO are summarised with respect to the HDL-C functionality.

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Omega-3 fatty acids and exercise: a review of their combined effects on body composition and physical performance

enriched with eicosapentaenoic acid (EPA) preserves lean body mass following esophageal cancer surgery: results of a double-blinded randomized controlled trial. Ann. Surg. 249:355-363. Saynor R., V. Derel, T. Gillott (1984) The long-term effect of dietary supplementation with fish lipid concentrate on serum lipids, bleeding time, platelets and angina. Atherosclerosis 50:3-10. Schacky, von C. (2007) n-3 PUFA in CVD: influence of cytokine polymorphism. Proc. Nutr. Soc. 66:166-170. Schacky

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