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Establishment of renal failure models by laparoscopy in bama pigs which underwent partial nephrectomy and radical contralateral nephrectomy

1997 64 1082 1086 12 Lin F., Cordes K., Li L., Hood L., Couser W.G., Shankland S.J., Igarashi P.: Hematopoietic stem cells contribute to the regeneration of renal tubules after renal ischemia-reperfusion injury in mice. J Am Soc Nephrol 2003, 14, 1188–1199. 12707389 10.1097/01.ASN.0000061595.28546.A0 Lin F. Cordes K. Li L. Hood L. Couser W.G. Shankland S.J. Igarashi P. Hematopoietic stem cells contribute to the regeneration of renal tubules after renal ischemia-reperfusion injury in mice J Am Soc Nephrol 2003 14 1188

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Alteration in Prooxidant-Antioxidant Balance Associated with Selenium Concentration in Patients with Congenital Hypothyroidism

. Parascandolo A, Rappa F, Cappello F, Kim J, Cantu DA, Chen H, et al. Extracellular superoxide dismutase expression in papillary thyroid cancer mesenchymal stem/stromal cells modulates cancer cell growth and migration. Sci Rep 2017; 7: 41416. 19. Ventura M, Melo M, Carrilho F. Selenium and thyroid disease: From pathophysiology to treatment. Int J Endo - crinol 2017; 2017: 1297658. 20. Reddy VS, Gouroju S, Suchitra MM, Suresh V, Sachan A, Srinivasa Rao PV, et al. Antioxidant defense in overt and subclinical hypothyroidism. Horm Metab Res 2013

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Problems of Cancer Treatment. Part 2. Treatment Based on Modification of Anticancer Immunological Responses in Therapy

-mesenchymal transition and a cancer stem cell-like phenotype in human colorectal cancer cells. Cancer Med. 2012; 1: 5-16 [22] Fridman WH, Remark R, Goc J, Giraldo NA, Becht E, Hammond SA, Damotte D, Dieu-Nosjean MC, Sautes-Fridman C. The immune microenvironment: a major player in human cancers. Int.Arch. Allergy Immunol. 2014; 164: 13-26 [23] Frydecka I, Kosmaczewska A, Bocko D, Ciszak L, Wolowiec D, Kuliczkowski K, Kochanowska I. Alterations of the expression of T-cell-related costimulatory CD28 and downregulatory CD152 (CTLA-4) molecules in patients

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The Most Important Transcriptional Factors of Osteoblastogenesis

Res 2009; 24: 1055-1065 [4] BESSA PC, CASAL M, REIS RL. Bone morphogenetic proteins in tissue engineering: the road from laboratory to clinic, part II (BMP delivery). J Tissue Eng Regen Med 2008; 2: 81-96. [5] BESSA PC, CASAL M, REIS RL. Bone morphogenetic proteins in tissue engineering: the road from the laboratory to the clinic, part I (basic concepts). J Tissue Eng Regen Med 2008; 2: 1-13. [6] BOBIS S, JAROCHA D, MAJKA M. Mesenchymal stem cells: characteristics and clinical applications. Folia Histochem Cytobiol

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Therapeutic Aspects of Catechin and Its Derivatives – An Update

(2017) Catechins activate muscle stem cells by Myf5 induction and stimulate muscle regeneration. Biochem Biophys Res Commun. 489(2):142-148. 22. Kimura-Ohba S, Yang Y (2016) Oxidative DNA Damage Mediated by Intranuclear MMP Activity Is Associated with Neuronal Apoptosis in Ischemic Stroke. Oxid. Med. Cell. Longev. 23. Koo SI, Noh SK (2007) Green Tea as Inhibitor of the Intestinal Absorption of Lipids: Potential Mechanism for its Lipid-Lowering Effect. J Nutr Biochem. 18(3):179-183. 24. Li F, Jin H, Xiao J, Yin

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Positive Regulation of Macromolecule Metabolic Process Belongs to the Main Mechanisms Crucial for Porcine Oocytes Maturation

. Two adjacent promoter elements mediate nerve growth factor activation of the c-fos gene and bind distinct nuclear complexes. Proc Nat Acad Sci. 1988; 85: 9474-9478. [40] Wang Z-Q, Grigoriadis AE, Mohle-Steinlein U, Wagner EF. A novel target cell for c-fos-induced oncogenesis: development of chondrogenictumours in embryonic stem cell chimeras. EMBO J. 1991; 10: 2437-2450. [41] Yam JWP, Jin D-Y, So CW, Chan LC. Identification and characterization of EBP, a novel EEN binding protein that inhibits Ras signaling and is recruited into the

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Participation of Membrane Nanotubes in Intercellular Communication

] Cselenyak A, Pankkotai E, Horvath E, Kiss L, Lacza Z. Mesenchymal stem cells rescue cardiomyoblasts from cell death in an in vitro ischemia model via direct cell-to-cell connections. BMC Cell Biol 2010; 11: 29. [13] Daubeuf S, Aucher A, Bordier Ch, Salles A, Serre L, Gaibelet G, Faye J-Ch, Favre G, Joly E, Hudrisier D. Preferential transfer of certain plasma membrane proteins onto T and B cells by trogocytosis. PLoS ONE 2010; 5: e8716. [14] Domhan S, Ma L, Tai A, Anaya Z, Beheshti A, Zeier M, Hlatky L, Abdollah I. Intercellular

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ARTs in wild felid conservation programmes in Poland and in the world

cryopreservation, and heterologous in vitro fertilization in the bobcat Lynx rufus Theriogenology 2009 72 341 52 9 Gómez M.C., Pope C.E., Giraldo A., Lyons L.A., Harris R.F., King A.L., Cole A., Godke R.A., Dresser B.L.: Birth of African wildcat cloned kittens born from domestic cats. Clon Stem Cells 2004, 6, 247–258. 10.1089/clo.2004.6.247 Gómez M.C. Pope C.E. Giraldo A. Lyons L.A. Harris R.F. King A.L. Cole A. Godke R.A. Dresser B.L. Birth of African wildcat cloned kittens born from domestic cats Clon Stem Cells 2004 6

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Clinical Utility of Survivin (BIRC5), Novel Cardiac Biomarker, as a Prognostic Tool Compared to High-Sensitivity C-Reactive Protein, Heart-Type Fatty Acid Binding Protein and Revised Lee Score in Elderly Patients Scheduled for Major Non-Cardiac Surgery: A Prospective Pilot Study

with the risk of coronary artery disease. J Med Biochem 2017; 36: 251–8. 39. Santini D, Abbate A, Scarpa S, Vasaturo F, Biondi-Zoccai GG, Bussani R, et al. Surviving acute myocardial infarction: survivin expression in viable cardiomyocytes after infarction. J Clin Pathol 2004; 57: 1321–4. 40. Ho YS, Tsai WH, Lin FC, Huang WP, Lin LC, Wu SM, et al. Cardioprotective actions of TGF RI inhibition through stimulating autocrine/paracrine of survivin and inhibiting Wnt in cardiac progenitors. Stem Cell 2015; 34(2): 445–55. 41. Lee BS, Kim SH, Jin T, Choi

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Application of targeted next generation sequencing for the mutational profiling of patients with acute lymphoblastic leukemia

Glycine at G48, in KRAS gene affecting Glycine at positions G12 and G13, and in NRAS gene the same amino-acid at position G12 and Alanine at A59. Identified mutations were mutually exclusive. This observation is in accordance with the traditional concept according to which only one mutation in each pathway is sufficient for disease development. In one model of ALL genesis, it was suggested that G12D variant in KRAS is a first genetic event responsible for malignant transformation of hematopoietic stem cells ( 26 ). Still, the majority of studies have focused on

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