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Epidemiology and Natural History of Nafld/Epidemiologija I Prirodna Istorija Nealkoholne Masne Jetre

adults: A systematic review. J Hepatol 2011; 56: 255-66. 7. Socha P, Horvath A, Vajro P, Dziechciarz P, Dhawan A, Szajewska H. Pharmacological interventions for nonalcoholic fatty liver disease in adults and in children: a systematic review. J Pediatr Gastroenterol Nutr 2009; 48: 587-96. 8. Bellentani S, Scaglioni F, Marino M, Bedogni G. Epide - miology of Fatty Liver Disease. Dig Dis 2010; 28: 155-61. 9. Browning JD, Szczepaniak LS, Dobbins R, Nuremberg P, Horton JD, Cohen JC, et al. Prevalence of hepatic steatosis in

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Factors That Influence the Virological Response in Patients with Chronic Hepatitis C Treated with Pegylated Interferon and Ribavirin

. 2011;54(4):1433-44. 27. Izumi N, Asahina Y, Kurosaki M. Predictors of Virological Response to a Combination Therapy with Pegylated Interferon Plus Ribavirin Including Virus and Host Factors. Hepatitis Research and Treatment. 2010;2010:703602. 28. Patton HM, Patel K, Behling C, et al. The impact of steatosis on disease progression and early and sustained treatment response in chronic hepatitis C patients. J Hepatol 2004;40:484-490. 29. Fierro NA, Gonzalez-Aldaco K, Torres-Valadez R, et al. Immunologic, metabolic and

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in PRILOZI
Liver disease symptoms in non-alcoholic fatty liver disease and small intestinal bacterial overgrowth

., DESCHENES M. Antioxidant therapy in nonalcoholic steatohepatitis . Hepat Res Treat. 2012; 22 : 947575. 7. VIGANÒ M., VERGANI A., TROMBINI P., POZZI M., PALEARI F., PIPERNO A. Insulin resistance influences iron metabolism and hepatic steatosis in type II diabetes. Gastroenterology. 2000; 118 (5):986-7. 8. COHEN B., NOVICK D., RUBINSTEIN M. Modulation of insulin activities by leptin. Science. 1996; 274 (5290):1185. 9. PAPPO I., BERCOVIER H., BERRY E., GALLILLY R., FEIGIN E., FREUND HR. Antitumor necrosis factor antibodies reduce hepatic steatosis

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Histopathological Changes in Rat Liver After A Single High Dose of Aluminium

Histopathological Changes in Rat Liver After A Single High Dose of Aluminium

Aluminium (Al) exposure may affect the liver of experimental animals. This investigation aimed at evaluating morphological changes in rat liver after a single high dose of Al (as metallic powder suspension). A total of forty female Wistar rats were divided in one exposed and one control group, 20 rats each. The exposed rats received 0.5 mL of sterile physiological suspension of fine Al powder in the concentration of 100 mg mL-1 intraperitoneally (50 mg Al per rat). After 7 weeks all animals were killed (by exsanguination from the abdominal aorta in ether anaesthesia). Liver aluminium was analysed using electrothermal atomic absorption spectrometry. For light microscopy the liver tissue was stained with hematoxylin and eosin, and for histochemical analysis with aurin threecarbocsillic acid (aluminon).

Liver Al level was markedly higher in the exposed (37.1 μg g-1) than in control rats (0.71 μg g-1). The exposed rats showed crystalloid Al inclusions in the capsular, subcapsular, and portal liver tissue. The basic liver structure remained intact. Slightly multiplied bile ductuli were found in 16 of 20 exposed and in 8 of 20 control rats. Three exposed rats had mycrovesicular steatosis. The peritoneum and Glisson's capsule showed strong macrophage infiltration and a foreign-body-like reaction with multiple giant macrophages containing Al crystalloid inclusions. Although this reaction was a defense against the metal, some Al passed this barrier and entered the liver tissue, exerting toxic effects in bile ductuli and hepatocytes.

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Factors Associated With Nonalcoholic Fatty Liver Disease in Obese Adolescents

(6): 1742-3. 10. Standard Egyptian Growth. Diabetes Endocrine Metabolism Pediatric Unit Cairo University Children's Hospital. [Last accessed on 2009 Aug 13, Last revised 2008 Nov 29]. Available from: http://www.dempuegypt.blogspot.com. 11. Ballentani S, Saccoccio G, Masutti F, et al. Prevalence of andrisk factors for hepatic steatosis in northern Italy. Ann Intern Med. 2000; 132:112-7. 12. Amarapurkar DN, Hashimoto E, Lesmana LA, Sollano JD, Chen PJ, Goh KL: How common is non-alcoholic fatty liver disease in the Asia

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Liver and Metabolic Diseases

Gastroenterol 21: 8293-8303, 2015. 24. Kim KH, Hong SP, Kim K, Park MJ, Kim KJ, Cheong J. HCV core protein induces hepatic lipid accumulation by activating SREBP1 and PPARgamma. Biochem Biophys Res Commun 355: 883-888, 2007. 25. Perlemuter G, Sabile A, Letteron P et al . Hepatitis C virus core protein inhibits microsomal triglyceride transfer protein activity and very low density lipoprotein secretion: a model of viral-related steatosis. FASEB J 16: 185-194, 2002. 26. Amako Y, Munakata T, Kohara M, Siddiqui A, Peers C, Harris M . Hepatitis C virus

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Non-alcoholic fatty pancreas disease – practices for clinicians

REFERENCES 1. ALEMPIJEVIC T., DRAGASEVIC S., ZEC S., POPOVIC D., MILOSAVLJEVIC T. Non-alcoholic fatty pancreas disease. Postgrad Med J 2017; 93 (1098):226-230. 2. SMITS M.M., VAN GEENEN E.J.M. The clinical significance of pancreatic steatosis. Nat Rev Gastroenterol Hepatol 2011; 8 (3): 169-177. 3. ROMANA B.S., CHELA H., DAILEY F.E., NASSIR F., TAHAN V. Non-alcoholic fatty pancreas disease (NAFPD). A silent spectator or the fifth component of metabolic syndrome? A literature review. Endocrine Metab Immune Disord – Drug Targets 2018; 18

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Rice Bran Oil and Pumpkin Seed Oil Alleviate Oxidative Injury and Fatty Liver in Rats Fed High Fructose Diet

protective and immunomodulatory effects of purslane, pumpkin and flax seeds on hypercholesterolemic rats. N. Am. J. Med. Sci., 2011, 3, 411-417. 12. Barbuio R., Milanski M., Bertolo M.B., Saad M.J., Vellosa L.A., Infl iximab reverses steatosis and improves insulin signal transduction in liver of rats fed a high-fat diet. J. Endocrinol., 2007, 194, 539-550. 13. Basciano H., Federico L., Adeli K., Fructose, insulin resistance, and metabolic dyslipidemia. Nutr. Metab. (Lond.), 2005, 2, 5-19. 14. Bray G.A., Fructose and risk of

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Elevated adiponectin is associated with poor outcome in children with biliary atresia

:364-70. 16. Arano T, Nakagawa H, Tateishi R, Ikeda H, Uchino K, Enooku K, et al. Serum level of adiponectin and the risk of liver cancer development in chronic hepatitis C patients. Int J Cancer. 2010;129:2226-35. 17. Baranova A, Jarrar MH, Stepanova M, Johnson A, Rafiq N, Gramlich T, et al. Association of serum adipocytokines with hepatic steatosis and fibrosis in patients with chronic hepatitis C. Digestion. 2011; 83: 32-40. 18. Latif HA, Assal HS, Mahmoud M, Rasheed WI. Role of serum adiponectin level in the development of liver cirrhosis

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Thirty years in hemostasis research in Cluj Napoca

References 1. Brudaşcă I, Cucuianu M. Pathogenic role of abnormal fatty acids and adipokines in the portal flow. Relevance for metabolic syndrome, hepatic steatosis and steatohepatitis. Rom J Intern Med. 2007;45(2):149-57. 2. Cucuianu M, Brudaşcă I. Coagulation factor XIII, impaired fibrinolysis and cardiovascular disease. Rev Romana Med Lab. 2011;19(2):119-27. 3. Brudaşcă I, Cucuianu M. Anticoagulant mechanisms are modulated by vascular endothelial cells. Rev Romana Med Lab. 2010;18(3):7-14. 4

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